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当心理社会解释增加精神问题污名时?自我报告和内隐证据。

When do psychosocial explanations of psychiatric problems increase stigma? Self-report and implicit evidence.

机构信息

Psychology Department, St. John Fisher College, 3690 East Avenue, Rochester, NY, 14618, USA.

出版信息

J Behav Ther Exp Psychiatry. 2019 Sep;64:15-21. doi: 10.1016/j.jbtep.2019.01.007. Epub 2019 Jan 28.

Abstract

BACKGROUND AND OBJECTIVES

Biomedical explanations of psychiatric problems, compared to psychosocial explanations, may amplify psychiatric stigma. One limitation of existing research is the measurement of almost exclusively self-reported stigma. This study evaluated the stigma-related effects of biomedical versus psychosocial explanations of schizophrenia using conventional self-report and two other measurement approaches that may tap more deeply held attitudes.

METHODS

One hundred three undergraduates listened to a vignette describing a man with (1) schizophrenia of biomedical origin, (2) schizophrenia of psychosocial origin, or (3) diabetes. They then completed an Implicit Association Test, conventional self-report stigma measures, and projected other measures that captured perceptions of most other people's likely impressions.

RESULTS

Participants were more likely to attribute stigmatizing views to others compared to themselves. The projected other measurement, but not the conventional self-report measurement, predicted implicit attitudes. We obtained no evidence that the psychosocial causal explanation of schizophrenia led to decreased stigma compared to the biomedical causal explanation. In fact, the psychosocial causal explanation increased stereotyped attitudes.

LIMITATIONS

The absence of a schizophrenia control group complicates interpretation of biomedical versus psychosocial group comparisons.

CONCLUSIONS

Further research is needed to evaluate discrepancies between the present findings and other published evidence pertaining to psychosocial causal explanations of psychiatric problems.

摘要

背景和目的

与心理社会解释相比,生物医学对精神问题的解释可能会加剧精神疾病污名化。现有研究的一个局限性在于几乎完全依赖自我报告来衡量污名化。本研究使用传统的自我报告和另外两种可能更深入地反映态度的测量方法,评估了精神分裂症的生物医学和心理社会解释与污名相关的影响。

方法

103 名本科生听取了一个描述一个患有(1)生物医学起源的精神分裂症、(2)心理社会起源的精神分裂症或(3)糖尿病的男子的案例。然后,他们完成了内隐联想测试、传统的自我报告污名测量,以及捕捉大多数其他人可能印象的投射其他测量。

结果

与自我相比,参与者更有可能将污名化的观点归因于他人。投射的其他测量方法,但不是传统的自我报告测量方法,预测了内隐态度。我们没有证据表明与生物医学病因解释相比,精神分裂症的心理社会病因解释会导致污名化减少。事实上,心理社会病因解释增加了刻板印象。

局限性

缺乏精神分裂症对照组使得生物医学与心理社会组比较的解释变得复杂。

结论

需要进一步研究,以评估本研究结果与其他关于精神问题的心理社会病因解释的已发表证据之间的差异。

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