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Ectopic ATP synthase β subunit proteins on human leukemia cell surface interact with platelets by binding glycoprotein IIb.

作者信息

Wang Ting, Shen Ying, Li Yuanyuan, Wang Beibei, Wang Benfang, Wu Depei, Ruan Changgeng, Wang Yiqiang

机构信息

MOH Key Lab of Thrombosis and Hemostasis, Collaborative Innovation Center of Hematology, Jiangsu Institute of Hematology, The First Affiliated Hospital of Soochow University, Medical College, Soochow University, Suzhou.

Center for Informational Biology, School of Life Science and Technology, University of Electronic Science and Technology of China, Chengdu and.

出版信息

Haematologica. 2019 Aug;104(8):e364-e368. doi: 10.3324/haematol.2019.216390. Epub 2019 Feb 7.

DOI:10.3324/haematol.2019.216390
PMID:30733262
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC6669168/
Abstract
摘要

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Ectopic ATP synthase β subunit proteins on human leukemia cell surface interact with platelets by binding glycoprotein IIb.人白血病细胞表面的异位ATP合酶β亚基蛋白通过结合糖蛋白IIb与血小板相互作用。
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2
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本文引用的文献

1
Systematic review of plasma-membrane ecto-ATP synthase: A new player in health and disease.系统综述血浆膜外三磷酸腺苷合酶:健康与疾病的新角色。
Exp Mol Pathol. 2018 Feb;104(1):59-70. doi: 10.1016/j.yexmp.2017.12.006. Epub 2018 Jan 2.
2
New Concepts and Mechanisms of Platelet Activation Signaling.血小板激活信号传导的新概念与机制
Physiology (Bethesda). 2017 Mar;32(2):162-177. doi: 10.1152/physiol.00020.2016.
3
[Platelets Decrease the Sensitivity of Leukemia Cell L1210 to Multiple Drugs via Activiting The AKT and ERK Signalling Pathway].[血小板通过激活AKT和ERK信号通路降低白血病细胞L1210对多种药物的敏感性]
Zhongguo Shi Yan Xue Ye Xue Za Zhi. 2016 Oct;24(5):1489-1494. doi: 10.7534/j.issn.1009-2137.2016.05.037.
4
Hyper-recovery of platelets after induction therapy is a predictor of relapse-free survival in acute myeloid leukemia.诱导治疗后血小板的超恢复是急性髓系白血病无复发生存的一个预测指标。
Leuk Lymphoma. 2017 Jan;58(1):104-109. doi: 10.1080/10428194.2016.1190969. Epub 2016 Jun 7.
5
Prognostic value of complete remission with superior platelet counts in acute myeloid leukemia.急性髓系白血病中血小板计数较高时完全缓解的预后价值
J Community Support Oncol. 2016 Feb;14(2):66-71. doi: 10.12788/jcso.0231.
6
Mechanistic explanation for platelet contribution to cancer metastasis.血小板促进癌症转移的机制解释。
Thromb Res. 2014 May;133 Suppl 2:S149-57. doi: 10.1016/S0049-3848(14)50025-4.
7
The αIIb p.Leu841Met (Cab3(a+) ) polymorphism results in a new human platelet alloantigen involved in neonatal alloimmune thrombocytopenia.αIIb 上的 p.Leu841Met(Cab3(a+))多态性导致了一种新的人类血小板同种异体抗原,该抗原与新生儿同种免疫性血小板减少症有关。
Transfusion. 2013 Mar;53(3):554-63. doi: 10.1111/j.1537-2995.2012.03762.x. Epub 2012 Jun 28.
8
Structure of a complete integrin ectodomain in a physiologic resting state and activation and deactivation by applied forces.完整整合素胞外结构域在生理静息状态下的结构以及外力作用下的激活与失活
Mol Cell. 2008 Dec 26;32(6):849-61. doi: 10.1016/j.molcel.2008.11.018.
9
Platelet-induced inhibition of tumor cell growth.血小板诱导的肿瘤细胞生长抑制。
Thromb Res. 2008;123(2):324-30. doi: 10.1016/j.thromres.2008.06.021. Epub 2008 Aug 9.
10
Effects of normal platelets on proliferation and constitutive cytokine secretion by human acute myelogenous leukaemia blasts.正常血小板对人急性髓系白血病原始细胞增殖及组成性细胞因子分泌的影响。
Platelets. 1997;8(6):397-404. doi: 10.1080/09537109777087.