Urinary Concentration Ability: Time to Bring the Tubules to the Table.
作者信息
Wang Ke, Kestenbaum Bryan
机构信息
Kidney Research Institute, Division of Nephrology, University of Washington, Seattle, WA.
Kidney Research Institute, Division of Nephrology, University of Washington, Seattle, WA.
出版信息
Am J Kidney Dis. 2019 May;73(5):583-584. doi: 10.1053/j.ajkd.2019.01.019. Epub 2019 Mar 12.
Abstract
摘要
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本文引用的文献
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Urine Ammonium Predicts Clinical Outcomes in Hypertensive Kidney Disease.尿铵可预测高血压肾病的临床结局。
J Am Soc Nephrol. 2017 Aug;28(8):2483-2490. doi: 10.1681/ASN.2016101151. Epub 2017 Apr 6.
2
Urine osmolarity and risk of dialysis initiation in a chronic kidney disease cohort--a possible titration target?慢性肾脏病队列中尿渗透压与开始透析的风险——一个可能的滴定目标？
PLoS One. 2014 Mar 27;9(3):e93226. doi: 10.1371/journal.pone.0093226. eCollection 2014.
3
Measured GFR does not outperform estimated GFR in predicting CKD-related complications.测量肾小球滤过率并不优于估计肾小球滤过率预测 CKD 相关并发症。
J Am Soc Nephrol. 2011 Oct;22(10):1931-7. doi: 10.1681/ASN.2010101077. Epub 2011 Sep 15.
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Autosomal dominant polycystic kidney disease.常染色体显性多囊肾病
Lancet. 2007 Apr 14;369(9569):1287-1301. doi: 10.1016/S0140-6736(07)60601-1.
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High urine volume and low urine osmolality are risk factors for faster progression of renal disease.高尿量和低尿渗透压是肾病进展加快的危险因素。
Am J Kidney Dis. 2003 May;41(5):962-71. doi: 10.1016/s0272-6386(03)00193-8.
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Dissociation of glomerular filtration rate from tubulointerstitial fibrosis in experimental chronic cyclosporine nephropathy: role of sodium intake.实验性慢性环孢素肾病中肾小球滤过率与肾小管间质纤维化的分离：钠摄入的作用
J Am Soc Nephrol. 1993 Aug;4(2):214-21. doi: 10.1681/ASN.V42214.
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On the pathogenesis of the uremic state. An exposition of the "trade-off hypothesis".论尿毒症状态的发病机制。“权衡假说”阐释
N Engl J Med. 1972 May 18;286(20):1093-9. doi: 10.1056/NEJM197205182862009.
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Tubulointerstitial changes as a major determinant in the progression of renal damage.肾小管间质改变是肾损伤进展的主要决定因素。
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