Immunologic characterization of tumor-associated transplantation antigens on MM102 mammary tumor eliciting preferentially helper T cell activity. - Suppr | 超能文献

Immunologic characterization of tumor-associated transplantation antigens on MM102 mammary tumor eliciting preferentially helper T cell activity.

作者信息

Fujiwara H, Aoki H, Tsuchida T, Hamaoka T

出版信息

J Immunol. 1978 Oct;121(4):1591-5.

Abstract

摘要

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1

Immunologic characterization of tumor-associated transplantation antigens on MM102 mammary tumor eliciting preferentially helper T cell activity.

J Immunol. 1978 Oct;121(4):1591-5.

2

Preferential generation of killer or helper T-lymphocyte activity directed to the tumour-associated transplantation antigens.优先产生针对肿瘤相关移植抗原的杀伤性或辅助性T淋巴细胞活性。

Immunology. 1976 Aug;31(2):239-48.

3

Correlation of in vitro and in vivo studies of antigens relevant to the control of murine breast cancer.与小鼠乳腺癌控制相关抗原的体外和体内研究的相关性

Cancer Res. 1976 Feb;36(2 pt 2):739-47.

4

Characterization of cytotoxic effector cells in the mouse mammary tumor system.小鼠乳腺肿瘤系统中细胞毒性效应细胞的特征分析。

J Immunol. 1975 Jan;114(1 Pt 1):24-9.

5

Expression of differentiation antigens cross-reactive with Ly-6.2 alloantigens on MM102 mouse mammary tumor cells.

Gan. 1982 Oct;73(5):758-65.

6

Occurrence of shared nonviral tumor-associated transplantation-type antigens among C3H/He mammary carcinomas.

Cancer Res. 1984 May;44(5):1876-9.

7

In vitro detection of immune responses to MTV-induced mammary tumors: activity of spleen cell preparations from both MTV-free and MTV-infected mice.体外检测对MTV诱导的乳腺肿瘤的免疫反应：来自无MTV和感染MTV小鼠的脾细胞制剂的活性。

J Immunol. 1974 Feb;112(2):693-705.

8

Immunodominance in the T cell response to multiple non-H-2 histocompatibility antigens. IV. Partial tissue distribution and mapping of immunodominant antigens.T细胞对多种非H-2组织相容性抗原反应中的免疫显性。IV. 免疫显性抗原的部分组织分布及定位

J Immunol. 1987 Oct 1;139(7):2166-71.

9

Role of donor T cells in the induction and transfer of allograft tolerance.

Transplant Proc. 1987 Feb;19(1 Pt 1):446-7.

10

A "stealth effect": adenocarcinoma cells engineered to express TRAIL elude tumor-specific and allogeneic T cell reactions.一种“隐匿效应”：经基因工程改造以表达肿瘤坏死因子相关凋亡诱导配体（TRAIL）的腺癌细胞能逃避肿瘤特异性及同种异体T细胞反应。

J Immunol. 1999 Nov 1;163(9):4886-93.

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Generation of TSTA diversity. Looking for testable hypotheses.TSTA多样性的产生。寻找可检验的假设。

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