Yıldırım Şeyda, Menteş Jale, Barış Mine
Adıyaman University Training and Research Hospital, Ophthalmology Clinic, Adıyaman, Turkey
Ege University Faculty of Medicine, Department of Ophthalmology, İzmir, Turkey
Turk J Ophthalmol. 2019 Apr 30;49(2):109-113. doi: 10.4274/tjo.galenos.2018.79577.
In this case report we aimed to present the outer retinal structural changes secondary to vitreomacular traction (VMT). Outer retinal structural changes occurring secondary to VMT due to incomplete posterior vitreous detachment were described retrospectively with spectral-domain optical coherence tomography in 3 eyes of 3 patients. The patients ranged in age from 58 to 65 years and best corrected visual acuity in the 3 eyes was 4/10, 8/10, and 9/10. All of the patients were symptomatic and exhibited outer retinal microholes at the fovea extending from the retinal pigment epithelium to outer limiting membrane, with an overlying operculum on the detached posterior hyaloid membrane over the macula following spontaneous resolution of VMT. In the mean follow-up period of 32 months, the outer retinal microholes decreased in size but did not completely resolve. As demonstrated in these cases, VMT can cause small outer retinal layer defects without signs of full-thickness macular hole. These lesions can cause symptoms and affect visual function, and may be permanent structural changes.
在本病例报告中,我们旨在呈现继发于玻璃体黄斑牵引(VMT)的视网膜外层结构变化。对3例患者的3只眼进行了回顾性研究,采用频域光学相干断层扫描描述了因不完全性玻璃体后脱离继发VMT所导致的视网膜外层结构变化。患者年龄在58至65岁之间,3只眼的最佳矫正视力分别为4/10、8/10和9/10。所有患者均有症状,且在黄斑中心凹处可见从视网膜色素上皮延伸至外限制膜的视网膜外层微孔,在VMT自发缓解后,黄斑上方脱离的后玻璃体膜上有覆盖的盖膜。在平均32个月的随访期内,视网膜外层微孔尺寸减小但未完全消失。如这些病例所示,VMT可导致视网膜外层小层缺损,而无全层黄斑裂孔的体征。这些病变可引起症状并影响视觉功能,且可能是永久性的结构改变。