Heller U, Dodenhöft J
HNO. 1987 Feb;35(2):67-9.
Tubal insufficiency evoked by inflammation or mechanical obstruction due to enlarged adenoids plays an important role in the pathogenesis of chronic serous otitis media. Serous and mucous tympanic effusions in the presence of immature middle ear mucosa and pneumatisation follow. An adhesive process can be the result. Secretory outflow as well as constant pressure compensation between the tympanic cavity and the auditory meatus can only be achieved by a long-term tympanic drainage. Progression of pneumatisation is achieved by recovery of the middle ear mucosa and in most cases an adhesive process can be prevented.
由炎症或腺样体肿大引起的机械性阻塞所诱发的咽鼓管功能不全，在慢性浆液性中耳炎的发病机制中起重要作用。继而在中耳黏膜未成熟和气化存在的情况下出现浆液性和黏液性鼓室积液。可能会导致粘连过程。只有通过长期的鼓膜引流，才能实现鼓室的分泌物流出以及鼓室与外耳道之间的恒定压力补偿。通过中耳黏膜的恢复实现气化进展，并且在大多数情况下可以防止粘连过程。
