Hörsted-Bindslev P
Scand J Dent Res. 1987 Aug;95(4):347-55. doi: 10.1111/j.1600-0722.1987.tb01852.x.
Deep buccal cavities were cut in 72 incisors and canines in six young monkeys. The cavities were restored as follows: Group One: acid etching of the enamel followed by an intermediate layer of unfilled resin and a light-cured microfilled composite. Group Two: acid etching of the enamel followed by treatment of the dentin with Gluma Cleanser and Gluma Bond. The cavities were restored as in Group One. Group Three: same procedure as in Group Two except for application of a calcium hydroxide cement on the pulpal wall prior to acid etching. Pulp reactions, presence and location of bacteria were studied after 8 and 90 days. A significant association between presence of bacteria and moderate to severe inflammatory reactions were observed. Bacteria were only found in a very few cavities pretreated with Gluma Dentin Bond. In general, slight inflammatory changes prevailed in Groups Two and Three. When the pulpal wall was extremely thin a toxic effect of Gluma Dentin Bond was observed.
在六只幼年猴子的72颗切牙和尖牙上制备了深颊面洞。这些洞按以下方式修复：第一组：对釉质进行酸蚀，然后涂一层未填充树脂中间层，再用光固化微填料复合树脂修复。第二组：对釉质进行酸蚀，然后用Gluma清洁剂和Gluma粘结剂处理牙本质。洞的修复方法同第一组。第三组：除在酸蚀前于髓壁上应用氢氧化钙水门汀外，操作步骤同第二组。在8天和90天后研究牙髓反应、细菌的存在及位置。观察到细菌的存在与中度至重度炎症反应之间存在显著关联。仅在极少数用Gluma牙本质粘结剂预处理的洞中发现细菌。总体而言，第二组和第三组中主要是轻微的炎症变化。当髓壁极薄时，观察到Gluma牙本质粘结剂有中毒作用。
