下颌骨萎缩与代谢性骨质流失。内分泌学、放射学与组织形态计量学。
Mandibular atrophy and metabolic bone loss. Endocrinology, radiology and histomorphometry.
作者信息
Habets L L, Bras J, Borgmeyer-Hoelen A M
机构信息
Department of Oral and Maxillofacial Surgery, University of Amsterdam, The Netherlands.
出版信息
Int J Oral Maxillofac Surg. 1988 Jun;17(3):208-11. doi: 10.1016/s0901-5027(88)80035-3.
In 11 edentulous patients with a severe atrophy of the mandible and submitted for ridge augmentation, endocrinological, radiological and histomorphometrical studies were carried out. The results showed that metabolic bone loss, histologically in nearly all patients characterized as a disturbance in mineralization and endocrinologically in 55-73% of the patients as a secondary hyperfunction of the parathyroid glands, plays a rôle in the etiology of mandibular atrophy.
对11例下颌严重萎缩并接受牙槽嵴增高术的无牙颌患者进行了内分泌、放射学和组织形态计量学研究。结果表明，代谢性骨丢失在下颌萎缩的病因中起作用，组织学上几乎所有患者均表现为矿化障碍，内分泌学上55%-73%的患者表现为甲状旁腺继发性功能亢进。
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