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本文引用的文献

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HEREDITARY PARTIAL AGENESIS OF CORPUS CALLOSUM; BIOCHEMICAL AND PATHOLOGICAL STUDIES.遗传性胼胝体部分发育不全；生物化学与病理学研究。
Arch Neurol. 1964 Aug;11:198-208. doi: 10.1001/archneur.1964.00460200094009.
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Induction of EAE in mice with recombinant human MOG, and treatment of EAE with a MOG peptide.
J Neuroimmunol. 1997 May;75(1-2):169-73. doi: 10.1016/s0165-5728(97)00019-2.
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Peripheral nerve involvement in merosin-deficient congenital muscular dystrophy and dy mouse.外周神经受累于缺乏层黏连蛋白α2的先天性肌营养不良和dy小鼠。
Neuromuscul Disord. 1997 Jan;7(1):7-12. doi: 10.1016/s0960-8966(96)00402-6.
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Resolution of CNS lesions following treatment of experimental allergic encephalomyelitis in macaques with monoclonal antibody to the CD18 leukocyte integrin.
Mult Scler. 1997 Jan;2(6):259-66. doi: 10.1177/135245859700200601.
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Glycoproteins of myelin sheaths.髓鞘糖蛋白。
J Mol Neurosci. 1997 Feb;8(1):1-12. doi: 10.1007/BF02736858.
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The human reelin gene: isolation, sequencing, and mapping on chromosome 7.人类reelin基因：克隆、测序及定位于7号染色体
Genome Res. 1997 Feb;7(2):157-64. doi: 10.1101/gr.7.2.157.
7
Arg-Gly-Asp-Ser-selective adhesion and the stabilization of long-term potentiation: pharmacological studies and the characterization of a candidate matrix receptor.精氨酸-甘氨酸-天冬氨酸-丝氨酸选择性黏附与长时程增强的稳定：药理学研究及一种候选基质受体的特性分析
J Neurosci. 1997 Feb 15;17(4):1320-9. doi: 10.1523/JNEUROSCI.17-04-01320.1997.
8
Enhanced aggregation of beta-amyloid-containing peptides by extracellular matrix and their degradation by the 68 kDa serine protease prepared from human brain.细胞外基质增强含β-淀粉样肽的聚集及其被从人脑中制备的68 kDa丝氨酸蛋白酶降解的过程
Neurosci Lett. 1996 Dec 20;220(3):159-62. doi: 10.1016/s0304-3940(96)13235-3.
9
Cell surface amyloid beta-protein precursor colocalizes with beta 1 integrins at substrate contact sites in neural cells.细胞表面淀粉样β蛋白前体在神经细胞的底物接触位点与β1整合素共定位。
J Neurosci. 1997 Feb 1;17(3):1004-10. doi: 10.1523/JNEUROSCI.17-03-01004.1997.
10
Myelin-associated glycoprotein inhibits neurite/axon growth and causes growth cone collapse.髓鞘相关糖蛋白抑制神经突/轴突生长并导致生长锥塌陷。
J Neurosci Res. 1996 Nov 15;46(4):404-14. doi: 10.1002/(SICI)1097-4547(19961115)46:4<404::AID-JNR2>3.0.CO;2-K.

Cell Adhesion Molecules: Implications for Neurological Disease.

作者信息

Fields R Douglas

机构信息

Laboratory of Developmental Neurobiology, National Institutes of Health, NICHD, Bethesda, Maryland.

出版信息

Neuroscientist. 1998 Jan 1;4(1):4-8. doi: 10.1177/107385849800400107.

DOI:10.1177/107385849800400107
PMID:31467472
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC6714583/
Abstract
摘要