Yagishita Atsuhiko, Akiyoshi Kikou, Goya Masahiko, Hirao Kenzo
Department of Cardiovascular Medicine / Heart Rhythm Center, Tokyo Medical and Dental University, Tokyo, Japan.
J Cardiol Cases. 2019 May 8;20(3):81-83. doi: 10.1016/j.jccase.2019.04.006. eCollection 2019 Sep.
The mechanism of adenosine-sensitive atrial tachycardia (AT) originating near the His bundle region has been reported to be due to reentry, as indicated by entrainment pacing at a site remote from the 'entrance' to the slow conduction zone of the postulated reentrant circuit. We report a case of AT, in which the change in response to the entrainment pacing following radiofrequency applications at the 'entrance' to the slow conduction zone not only suggested that the mechanism of the AT was reentry, but also the size of the reentrant circuit. < The mechanism of adenosine-sensitive atrial tachycardia (AT) with an earliest atrial activation site near the His bundle has been reported to be due to reentry, but the size of the postulated reentrant circuit remains uncertain. This case suggested not only the mechanism of the AT was reentry, but also the size of the reentrant circuit localized near the His bundle based on the response to entrainment pacing after the radiofrequency applications at the 'entrance' to the slow conduction zone of the reentrant circuit.>.
据报道，起源于希氏束区域附近的腺苷敏感性房性心动过速（AT）的机制是折返，这一点通过在远离假定折返环路慢传导区“入口”处的部位进行拖带起搏得以证实。我们报告了一例房性心动过速病例，在对慢传导区“入口”进行射频消融后，拖带起搏反应的变化不仅提示房性心动过速的机制为折返，还提示了折返环路的大小。<据报道，最早心房激动部位靠近希氏束的腺苷敏感性房性心动过速（AT）的机制是折返，但假定折返环路的大小仍不确定。该病例不仅提示房性心动过速的机制为折返，还根据在折返环路慢传导区“入口”进行射频消融后对拖带起搏的反应，提示了位于希氏束附近的折返环路的大小。>
