Huber B, Melchers F
Eur J Immunol. 1979 Oct;9(10):827-9. doi: 10.1002/eji.1830091016.
B cell-defective (CBA/N X BALB/c)F1 male mice have 100 times less lipopolysaccharide-reactive and 10--100 times less lipoprotein- and Nocardia mitogen-reactive B cell precursors than F1 female mice with normal B cells.
B细胞缺陷的(CBA/N×BALB/c)F1雄性小鼠的脂多糖反应性B细胞前体比具有正常B细胞的F1雌性小鼠少100倍,脂蛋白和诺卡氏菌促细胞分裂剂反应性B细胞前体少10至100倍。