Declining Pulmonary Function in Interstitial Lung Disease Linked to Lymphocyte Dysfunction.
作者信息
Schott Cody A, Ascoli Christian, Huang Yue, Perkins David L, Finn Patricia W
机构信息
University of Illinois at Chicago College of MedicineChicago, Illinois.
出版信息
Am J Respir Crit Care Med. 2020 Mar 1;201(5):610-613. doi: 10.1164/rccm.201910-1909LE.
Abstract
摘要
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本文引用的文献
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Telomere Length and Use of Immunosuppressive Medications in Idiopathic Pulmonary Fibrosis.特发性肺纤维化中端粒长度与免疫抑制药物的使用。
Am J Respir Crit Care Med. 2019 Aug 1;200(3):336-347. doi: 10.1164/rccm.201809-1646OC.
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PD-1 up-regulation on CD4 T cells promotes pulmonary fibrosis through STAT3-mediated IL-17A and TGF-β1 production.PD-1 在 CD4 T 细胞上的上调通过 STAT3 介导的 IL-17A 和 TGF-β1 产生促进肺纤维化。
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Local and Systemic CD4 T Cell Exhaustion Reverses with Clinical Resolution of Pulmonary Sarcoidosis.局部和全身 CD4 T 细胞耗竭随肺结节病的临床缓解而逆转。
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A Circulating MicroRNA Signature Serves as a Diagnostic and Prognostic Indicator in Sarcoidosis.循环微小RNA特征作为结节病的诊断和预后指标
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Sphingosine-1-phosphate lyase is an endogenous suppressor of pulmonary fibrosis: role of S1P signalling and autophagy.鞘氨醇-1-磷酸酶是肺纤维化的内源性抑制剂：S1P 信号和自噬的作用。
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Blockade of the programmed death-1 pathway restores sarcoidosis CD4(+) T-cell proliferative capacity.程序性死亡-1通路的阻断可恢复结节病CD4(+) T细胞的增殖能力。
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Peripheral blood mononuclear cell gene expression profiles predict poor outcome in idiopathic pulmonary fibrosis.特发性肺纤维化患者外周血单个核细胞基因表达谱预测不良预后。
Sci Transl Med. 2013 Oct 2;5(205):205ra136. doi: 10.1126/scitranslmed.3005964.
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Reversal of global CD4+ subset dysfunction is associated with spontaneous clinical resolution of pulmonary sarcoidosis.CD4+ 亚群功能全球逆转与肺结节病自发临床缓解相关。
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Prednisone, azathioprine, and N-acetylcysteine for pulmonary fibrosis.泼尼松、硫唑嘌呤和 N-乙酰半胱氨酸治疗肺纤维化。
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