• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利:CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法,用中文像聊天一样搜索,搜遍4000万医学文献。AI智能推荐,让科研检索更轻松。

立即免费搜索

文件翻译

保留排版,准确专业,支持PDF/Word/PPT等文件格式,支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述,25分钟生成高质量综述,智能提取关键信息,辅助科研写作。

立即免费体验

相似文献

1
Declining Pulmonary Function in Interstitial Lung Disease Linked to Lymphocyte Dysfunction.间质性肺病中肺功能下降与淋巴细胞功能障碍有关。
Am J Respir Crit Care Med. 2020 Mar 1;201(5):610-613. doi: 10.1164/rccm.201910-1909LE.
2
Different activity of the biological axis VEGF-Flt-1 (fms-like tyrosine kinase 1) and CXC chemokines between pulmonary sarcoidosis and idiopathic pulmonary fibrosis: a bronchoalveolar lavage study.结节病与特发性肺纤维化之间生物轴VEGF-Flt-1(fms样酪氨酸激酶1)和CXC趋化因子的不同活性:一项支气管肺泡灌洗研究
Clin Dev Immunol. 2009;2009:537929. doi: 10.1155/2009/537929. Epub 2010 Feb 14.
3
Pulmonary sarcoidosis is associated with high-level inducible co-stimulator (ICOS) expression on lung regulatory T cells--possible implications for the ICOS/ICOS-ligand axis in disease course and resolution.肺结节病与肺调节性T细胞上高水平的诱导性共刺激分子(ICOS)表达相关——这可能对疾病进程和缓解过程中的ICOS/ICOS配体轴有影响。
Clin Exp Immunol. 2016 Feb;183(2):294-306. doi: 10.1111/cei.12715. Epub 2015 Nov 24.
4
T-bet Expression in Peripheral Th17.0 Cells Is Associated With Pulmonary Function Changes in Sarcoidosis.T 细胞转录因子在周围 Th17.0 细胞中的表达与结节病的肺功能变化有关。
Front Immunol. 2020 Jul 22;11:1129. doi: 10.3389/fimmu.2020.01129. eCollection 2020.
5
Aberrant populations of circulating T follicular helper cells and regulatory B cells underlying idiopathic pulmonary fibrosis.特发性肺纤维化患者循环滤泡辅助 T 细胞和调节性 B 细胞的异常。
Respir Res. 2019 Nov 6;20(1):244. doi: 10.1186/s12931-019-1216-6.
6
MRP14 is elevated in the bronchoalveolar lavage fluid of fibrosing interstitial lung diseases.MRP14 在纤维化性间质性肺疾病的支气管肺泡灌洗液中升高。
Clin Exp Immunol. 2010 Aug;161(2):342-7. doi: 10.1111/j.1365-2249.2010.04181.x. Epub 2010 Jun 9.
7
Clinical evaluation of lymphocyte sub-populations and oxygen radical production in sarcoidosis and idiopathic pulmonary fibrosis.结节病和特发性肺纤维化中淋巴细胞亚群及氧自由基产生的临床评估
Respir Med. 1994 Jan;88(1):55-64. doi: 10.1016/0954-6111(94)90175-9.
8
Endpoints in sarcoidosis: More like IPF or asthma?结节病的终点:更像特发性肺纤维化还是哮喘?
Respir Med. 2018 May;138S:S3-S4. doi: 10.1016/j.rmed.2017.11.010. Epub 2017 Nov 20.
9
Real-life experiences in a single center: efficacy of pirfenidone in idiopathic pulmonary fibrosis and fibrotic idiopathic non-specific interstitial pneumonia patients.单中心真实世界经验:吡非尼酮治疗特发性肺纤维化和特发性非特异性间质性肺炎纤维化患者的疗效。
Ther Adv Respir Dis. 2020 Jan-Dec;14:1753466620963015. doi: 10.1177/1753466620963015.
10
Polarization of ILC2s in peripheral blood might contribute to immunosuppressive microenvironment in patients with gastric cancer.ILC2s 在周围血液中的极化可能有助于胃癌患者的免疫抑制微环境。
J Immunol Res. 2014;2014:923135. doi: 10.1155/2014/923135. Epub 2014 Mar 4.

引用本文的文献

1
Peripheral Transcriptome of Severe Sarcoidosis Involves Dysregulation of Multiple Immunologic Systems.重症结节病的外周转录组涉及多个免疫系统的失调。
Am J Respir Cell Mol Biol. 2025 Jun;72(6):712-715. doi: 10.1165/rcmb.2024-0199LE.
2
The Relationship Between Differential Expression of Non-coding RNAs (TP53TG1, LINC00342, MALAT1, DNM3OS, miR-126-3p, miR-200a-3p, miR-18a-5p) and Protein-Coding Genes (PTEN, FOXO3) and Risk of Idiopathic Pulmonary Fibrosis.非编码RNA(TP53TG1、LINC00342、MALAT1、DNM3OS、miR-126-3p、miR-200a-3p、miR-18a-5p)与蛋白质编码基因(PTEN、FOXO3)的差异表达与特发性肺纤维化风险的关系
Biochem Genet. 2025 Jan 29. doi: 10.1007/s10528-024-11012-z.
3
The immune mechanisms of acute exacerbations of idiopathic pulmonary fibrosis.特发性肺纤维化急性加重的免疫机制
Front Immunol. 2024 Dec 16;15:1450688. doi: 10.3389/fimmu.2024.1450688. eCollection 2024.
4
Effects of Anti-Fibrotic Drugs on Transcriptome of Peripheral Blood Mononuclear Cells in Idiopathic Pulmonary Fibrosis.抗纤维化药物对特发性肺纤维化患者外周血单个核细胞转录组的影响。
Int J Mol Sci. 2024 Mar 28;25(7):3750. doi: 10.3390/ijms25073750.
5
Vaccination in the Era of Immunosuppression.免疫抑制时代的疫苗接种
Vaccines (Basel). 2023 Sep 1;11(9):1446. doi: 10.3390/vaccines11091446.
6
IKAROS in Acute Leukemia: A Positive Influencer or a Mean Hater?IKAROS 在急性白血病中的作用:正面影响者还是恶意攻击者?
Int J Mol Sci. 2023 Feb 7;24(4):3282. doi: 10.3390/ijms24043282.
7
Altered transcription factor targeting is associated with differential peripheral blood mononuclear cell proportions in sarcoidosis.转录因子靶向改变与结节病外周血单个核细胞比例的差异有关。
Front Immunol. 2022 Oct 13;13:848759. doi: 10.3389/fimmu.2022.848759. eCollection 2022.
8
Ikaros Proteins in Tumor: Current Perspectives and New Developments.肿瘤中的Ikaros蛋白:当前观点与新进展
Front Mol Biosci. 2021 Dec 7;8:788440. doi: 10.3389/fmolb.2021.788440. eCollection 2021.
9
Update in Interstitial Lung Disease 2020.2020年间质性肺疾病进展
Am J Respir Crit Care Med. 2021 Jun 1;203(11):1343-1352. doi: 10.1164/rccm.202103-0559UP.
10
Unsupervised Clustering Reveals Sarcoidosis Phenotypes Marked by a Reduction in Lymphocytes Relate to Increased Inflammatory Activity on 18FDG-PET/CT.无监督聚类揭示了以淋巴细胞减少为特征的结节病表型与18FDG-PET/CT上炎症活动增加有关。
Front Med (Lausanne). 2021 Feb 24;8:595077. doi: 10.3389/fmed.2021.595077. eCollection 2021.

本文引用的文献

1
Telomere Length and Use of Immunosuppressive Medications in Idiopathic Pulmonary Fibrosis.特发性肺纤维化中端粒长度与免疫抑制药物的使用。
Am J Respir Crit Care Med. 2019 Aug 1;200(3):336-347. doi: 10.1164/rccm.201809-1646OC.
2
PD-1 up-regulation on CD4 T cells promotes pulmonary fibrosis through STAT3-mediated IL-17A and TGF-β1 production.PD-1 在 CD4 T 细胞上的上调通过 STAT3 介导的 IL-17A 和 TGF-β1 产生促进肺纤维化。
Sci Transl Med. 2018 Sep 26;10(460). doi: 10.1126/scitranslmed.aar8356.
3
Local and Systemic CD4 T Cell Exhaustion Reverses with Clinical Resolution of Pulmonary Sarcoidosis.局部和全身 CD4 T 细胞耗竭随肺结节病的临床缓解而逆转。
J Immunol Res. 2017;2017:3642832. doi: 10.1155/2017/3642832. Epub 2017 Nov 6.
4
A Circulating MicroRNA Signature Serves as a Diagnostic and Prognostic Indicator in Sarcoidosis.循环微小RNA特征作为结节病的诊断和预后指标
Am J Respir Cell Mol Biol. 2018 Jan;58(1):40-54. doi: 10.1165/rcmb.2017-0207OC.
5
Sphingosine-1-phosphate lyase is an endogenous suppressor of pulmonary fibrosis: role of S1P signalling and autophagy.鞘氨醇-1-磷酸酶是肺纤维化的内源性抑制剂:S1P 信号和自噬的作用。
Thorax. 2015 Dec;70(12):1138-48. doi: 10.1136/thoraxjnl-2014-206684. Epub 2015 Aug 18.
6
Blockade of the programmed death-1 pathway restores sarcoidosis CD4(+) T-cell proliferative capacity.程序性死亡-1通路的阻断可恢复结节病CD4(+) T细胞的增殖能力。
Am J Respir Crit Care Med. 2014 Sep 1;190(5):560-71. doi: 10.1164/rccm.201401-0188OC.
7
Peripheral blood mononuclear cell gene expression profiles predict poor outcome in idiopathic pulmonary fibrosis.特发性肺纤维化患者外周血单个核细胞基因表达谱预测不良预后。
Sci Transl Med. 2013 Oct 2;5(205):205ra136. doi: 10.1126/scitranslmed.3005964.
8
Reversal of global CD4+ subset dysfunction is associated with spontaneous clinical resolution of pulmonary sarcoidosis.CD4+ 亚群功能全球逆转与肺结节病自发临床缓解相关。
J Immunol. 2013 Jun 1;190(11):5446-53. doi: 10.4049/jimmunol.1202891. Epub 2013 Apr 29.
9
Peripheral blood gene expression as a novel genomic biomarker in complicated sarcoidosis.外周血基因表达作为复杂结节病的新型基因组生物标志物。
PLoS One. 2012;7(9):e44818. doi: 10.1371/journal.pone.0044818. Epub 2012 Sep 12.
10
Prednisone, azathioprine, and N-acetylcysteine for pulmonary fibrosis.泼尼松、硫唑嘌呤和 N-乙酰半胱氨酸治疗肺纤维化。
N Engl J Med. 2012 May 24;366(21):1968-77. doi: 10.1056/NEJMoa1113354. Epub 2012 May 20.

Declining Pulmonary Function in Interstitial Lung Disease Linked to Lymphocyte Dysfunction.

作者信息

Schott Cody A, Ascoli Christian, Huang Yue, Perkins David L, Finn Patricia W

机构信息

University of Illinois at Chicago College of MedicineChicago, Illinois.

出版信息

Am J Respir Crit Care Med. 2020 Mar 1;201(5):610-613. doi: 10.1164/rccm.201910-1909LE.

DOI:10.1164/rccm.201910-1909LE
PMID:31661301
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7047459/
Abstract
摘要