Merchut M P, Gupta S R, Naheedy M H
Department of Neurology, Loyola University Medical Center, Maywood, IL 60153.
Stroke. 1988 Oct;19(10):1239-42. doi: 10.1161/01.str.19.10.1239.
We retrospectively studied 46 patients with symptomatic retinal artery occlusion and assessed the pattern and extent of carotid artery disease ipsilateral to the retinal artery occlusion. Ipsilateral internal carotid artery atherosclerotic lesions were virtually limited to the cervical arterial segment; 50% of such lesions were plaques or stenoses of less than or equal to 60%, whereas 15% of the angiograms were normal. No clinical features were significantly associated with a flow-limiting carotid stenosis of greater than 60%. Contrary to previous reports, the type of retinal artery occlusion, whether branch or central artery occlusion, was not predictive of severe underlying carotid stenosis or occlusion. Likely mechanisms of retinal artery occlusion include in situ thrombosis and emboli from carotid, and possibly cardiac, sources. Extension of thrombus from an occluded carotid artery into the ophthalmic artery did not appear to be a mechanism of retinal artery occlusion.
我们回顾性研究了46例有症状的视网膜动脉阻塞患者，并评估了视网膜动脉阻塞同侧颈动脉疾病的类型和范围。同侧颈内动脉粥样硬化病变实际上局限于颈动脉颈段；50%的此类病变为斑块或狭窄程度小于或等于60%，而15%的血管造影结果正常。没有临床特征与大于60%的限流性颈动脉狭窄显著相关。与之前的报道相反，视网膜动脉阻塞的类型，无论是分支动脉阻塞还是中央动脉阻塞，都不能预测严重的潜在颈动脉狭窄或阻塞。视网膜动脉阻塞的可能机制包括原位血栓形成以及来自颈动脉甚至可能来自心脏的栓子。血栓从阻塞的颈动脉延伸至眼动脉似乎不是视网膜动脉阻塞的机制。
