Korochkin I M, Kartelishev A V, Leshakov S Iu, Kapustina G M, Vernekina N S
Kardiologiia. 1988 Aug;28(8):72-5.
Some pathochemical mechanisms of coronary disease aggravation in the course of laser therapy were assessed by means of the measurement of erythrocyte membrane lipids and phospholipids as well as plasma alpha-tocopherol and diene conjugate levels. The aggravation was associated with an activation of metabolic processes aiming to produce regeneration of membrane structures. The regeneration takes place in the presence of chronic deficiency of total phospholipids and inadequate antioxidant protection of biomembranes, therefore complete stabilization and normalization of their composition and activity is impossible.
通过测量红细胞膜脂质和磷脂以及血浆α-生育酚和二烯结合物水平，评估了激光治疗过程中冠心病加重的一些病理化学机制。病情加重与旨在产生膜结构再生的代谢过程激活有关。再生发生在总磷脂长期缺乏和生物膜抗氧化保护不足的情况下，因此其组成和活性不可能完全稳定和正常化。
