Oshina Masahito, Segawa Tomohide, Oshima Yasushi, Tanaka Sakae, Inanami Hirohiko
Department of Orthopedic Surgery, Inanami Spine and Joint Hospital, 3-17-5, Higashishinagawa, Shinagawa-Ku.
Department of Orthopedic Surgery, The University of Tokyo Hospital 7-3-1, Hongo, Bunkyo-Ku, Tokyo, Japan.
Medicine (Baltimore). 2020 Jan;99(3):e18817. doi: 10.1097/MD.0000000000018817.
Although C5 palsy is a common complication of cervical spine surgery, its cause has not been confirmed. There are various hypotheses for its mechanism, including spinal cord impairment and nerve involvement. Therefore, prophylactic foraminotomy is one of the methods recommended for preventing C5 palsy. However, we describe a patient who experienced C5 palsy after microendoscopic foraminotomy between the left C5/6 and C6/7 levels.
A 43-year-old man presented with a 14-month history of progressive numbness in the left upper limb. We performed microendoscopic left foraminal decompressions at the C5/6/7 levels to treat the left C6 and C7 radiculopathy. On the postoperative day 1, we observed weak motor strength of the left deltoid, left biceps, and left forearm pronator, while the motor strength of the other muscles was normal.
C5 palsy following C5/6/7 left foraminotomy.
Follow-up rehabilitation with muscle strength training and range of motion training.
The patient recovered his motor strength completely within 3 months postoperatively.
In this case, the C5 palsy could not be adequately explained by the theory of nerve root impingement or disruption in blood circulation following spinal cord decompression. We hypothesized that the patient had drill heat-induced C5 palsy. Regarding the C5 palsy without C5 nerve root decompression, we hypothesize that the C5 palsy in C5/6/7 foraminotomy could be related to variations in the formation of the brachial plexus. Prophylactic foraminotomy for cervical posterior surgery should be performed with care, limiting its use in patients who are at a risk of developing C5 palsy because the prophylactic procedure can cause C5 palsy. We must also consider that even without decompression at the C4/5 level, there is a possibility of C5 palsy occurring.
尽管C5麻痹是颈椎手术常见的并发症,但其病因尚未得到证实。关于其机制有多种假说,包括脊髓损伤和神经受累。因此,预防性椎间孔切开术是推荐用于预防C5麻痹的方法之一。然而,我们描述了一名患者,其在左侧C5/6和C6/7节段进行显微内镜下椎间孔切开术后出现了C5麻痹。
一名43岁男性,有14个月的左上肢进行性麻木病史。我们在C5/6/7节段进行了显微内镜下左侧椎间孔减压术,以治疗左侧C6和C7神经根病。术后第1天,我们观察到左侧三角肌、左侧肱二头肌和左侧前臂旋前肌肌力减弱,而其他肌肉肌力正常。
C5/6/7左侧椎间孔切开术后C5麻痹。
进行肌肉力量训练和关节活动范围训练的随访康复。
患者术后3个月内完全恢复了肌力。
在本病例中,神经根受压理论或脊髓减压后血液循环中断理论均无法充分解释C5麻痹。我们推测该患者为钻磨热所致C5麻痹。对于未进行C5神经根减压的C5麻痹,我们推测C5/6/7椎间孔切开术中的C5麻痹可能与臂丛神经形成变异有关。颈椎后路手术的预防性椎间孔切开术应谨慎进行,限制其在有发生C5麻痹风险的患者中的使用,因为预防性手术可能导致C5麻痹。我们还必须考虑到,即使未在C4/5节段进行减压,也有可能发生C5麻痹。