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Letter to the Editor: "Histone Deacetylase 7 Inhibition in a Murine Model of Gram-Negative Pneumonia-Induced Acute Lung Injury" Shock 53:344-351, 2020.致编辑的信:“组蛋白去乙酰化酶7抑制在革兰氏阴性菌肺炎诱导的急性肺损伤小鼠模型中的作用”,《休克》第53卷:344 - 351页,2020年。
Shock. 2020 Mar;53(3):375. doi: 10.1097/SHK.0000000000001430.
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Trichostatin A attenuates ventilation-augmented epithelial-mesenchymal transition in mice with bleomycin-induced acute lung injury by suppressing the Akt pathway.曲古抑菌素A通过抑制Akt信号通路减轻博来霉素诱导的急性肺损伤小鼠中通气增强的上皮-间质转化。
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Zinc-Dependent Histone Deacetylases in Lung Endothelial Pathobiology.锌依赖的组蛋白去乙酰化酶在肺血管内皮病理生物学中的作用
Biomolecules. 2024 Jan 23;14(2):140. doi: 10.3390/biom14020140.

本文引用的文献

1
Histone Deacetylase 7 Inhibition in a Murine Model of Gram-Negative Pneumonia-Induced Acute Lung Injury.组蛋白去乙酰化酶7抑制在革兰氏阴性菌肺炎诱导的急性肺损伤小鼠模型中的作用
Shock. 2020 Mar;53(3):344-351. doi: 10.1097/SHK.0000000000001372.
2
Histone Deacetylase Inhibition Protects Mice Against Lethal Postinfluenza Pneumococcal Infection.组蛋白去乙酰化酶抑制可保护小鼠免受致命的流感后肺炎球菌感染。
Crit Care Med. 2016 Oct;44(10):e980-7. doi: 10.1097/CCM.0000000000001821.
3
Autotaxin is induced by TSA through HDAC3 and HDAC7 inhibition and antagonizes the TSA-induced cell apoptosis.自分泌酶受 TSA 通过抑制 HDAC3 和 HDAC7 诱导,并拮抗 TSA 诱导的细胞凋亡。
Mol Cancer. 2011 Feb 12;10:18. doi: 10.1186/1476-4598-10-18.
4
HDAC family: What are the cancer relevant targets?组蛋白去乙酰化酶家族:与癌症相关的靶点有哪些?
Cancer Lett. 2009 May 8;277(1):8-21. doi: 10.1016/j.canlet.2008.08.016. Epub 2008 Sep 27.
5
Structures of a histone deacetylase homologue bound to the TSA and SAHA inhibitors.与TSA和SAHA抑制剂结合的组蛋白脱乙酰酶同源物的结构。
Nature. 1999 Sep 9;401(6749):188-93. doi: 10.1038/43710.

Letter to the Editor: "Histone Deacetylase 7 Inhibition in a Murine Model of Gram-Negative Pneumonia-Induced Acute Lung Injury" Shock 53:344-351, 2020.

作者信息

Verin Alexander D

机构信息

Vascular Biology Center Medical College of Georgia at Augusta University Augusta, Georgia.

出版信息

Shock. 2020 Mar;53(3):375. doi: 10.1097/SHK.0000000000001430.

DOI:10.1097/SHK.0000000000001430
PMID:32044849
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC9970038/
Abstract
摘要