离心性肌肉收缩导致的组织降解标志物变化及疼痛主观报告
CHANGES IN TISSUE DEGRADATION MARKERS AND SUBJECTIVE REPORTS OF PAIN RESULTING FROM ECCENTRIC MUSCLE CONTRACTIONS.
作者信息
Geisler P R, Hackney A C, McMurray R G, Ainsworth B A
机构信息
Applied Physiology Laboratory, University of North Carolina, Chapel Hill NC, U.S.A.
出版信息
Biol Sport. 1996;13(1):13-20.
The delayed onset of muscular soreness (DOMS) following a heavy eccentric exercise was studied with the aim to verify the "muscular structure" and the "connective tissue" theories explaining the development of DOMS. Die responses of creatine kinase (CK; "structural theory" marker) and hydroxyproline (OHP; "connective tissue theory" marker), as well as level and location of the perceived soreness, were determined following eccentric exercise. Plasma CK activity was elevated 48 and 72 h after the acentric exercise compared with the pre-exercise values, while OHP concentrations remained unchanged. Examination of pain location reports revealed two groups of responders (distal vs. mid-point muscle pain). Distal pain responders were found to have significantly higher post-exercise CK activity than mid-point pain responders, the OHP levels being alike. These findings are supportive of the "muscular structure" theory for DOMS development. However, the non-uniform location for DOMS pain confounds the overall data interpretation.
为了验证解释延迟性肌肉酸痛（DOMS）产生的“肌肉结构”和“结缔组织”理论，对高强度离心运动后延迟性肌肉酸痛的情况进行了研究。在离心运动后，测定了肌酸激酶（CK；“结构理论”标志物）和羟脯氨酸（OHP；“结缔组织理论”标志物）的反应，以及感知到的酸痛程度和位置。与运动前的值相比，离心运动后48小时和72小时血浆CK活性升高，而OHP浓度保持不变。对疼痛位置报告的检查发现了两组反应者（远端肌肉疼痛与中点肌肉疼痛）。发现远端疼痛反应者运动后的CK活性明显高于中点疼痛反应者，而OHP水平相似。这些发现支持了DOMS产生的“肌肉结构”理论。然而，DOMS疼痛位置的不一致使得整体数据解释变得复杂。
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