Müller S C, Hsieh J T, Lue T F, Tanagho E A
Johannes Gutenberg University, Mainz, FRG.
Eur Urol. 1988;15(1-2):118-24. doi: 10.1159/000473410.
Castrated dogs (n = 3) need a much higher threshold level of energy to induce erection by electrical stimulation of the cavernous nerve than noncastrated animals (n = 24). In addition the resulting quality of erection, measured as maximal intracavernous pressure (pCC) versus peak systolic blood pressure (BP), was weaker in castrated dogs (pCC = 57% of BP on average) than in noncastrated dogs (pCC = 80% of BP on average). A high venous outflow from the corpora cavernosa in castrated dogs can also explain the shorter duration of erection. This experimental model excludes the interference of subjective factors, such as erotic stimuli and libido on erection, and it seems that androgen deficiency has a direct effect on the neurophysiology of the erectile tissues resulting in a higher tonus of the detumescence factors, which can be explained by an incomplete relaxation of the sinusoidal smooth muscle.
阉割犬（n = 3）通过海绵体神经电刺激诱导勃起所需的能量阈值水平比未阉割动物（n = 24）高得多。此外，以最大海绵体内压（pCC）与收缩压峰值（BP）衡量的勃起质量，在阉割犬中（平均pCC = BP的57%）比未阉割犬（平均pCC = BP的80%）弱。阉割犬海绵体的高静脉流出也可以解释勃起持续时间较短的原因。该实验模型排除了主观因素如性刺激和性欲对勃起的干扰，雄激素缺乏似乎对勃起组织的神经生理学有直接影响，导致消肿因子张力更高，这可以通过窦状平滑肌不完全松弛来解释。
