From Neutrophil Calcium Sensor to Myocardial Remodelling-A Double-Edged Pathway Guided by the Inflammasome.
作者信息
Scott Larry, Li Na
机构信息
Section of Cardiovascular Research, Department of Medicine, Baylor College of Medicine, Houston, Texas, USA; Department of Molecular Physiology and Biophysics, Baylor College of Medicine, Houston, Texas, USA; Cardiovascular Research Institute, Baylor College of Medicine, Houston, Texas, USA.
Section of Cardiovascular Research, Department of Medicine, Baylor College of Medicine, Houston, Texas, USA; Department of Molecular Physiology and Biophysics, Baylor College of Medicine, Houston, Texas, USA; Cardiovascular Research Institute, Baylor College of Medicine, Houston, Texas, USA.
出版信息
Can J Cardiol. 2020 Jun;36(6):813-815. doi: 10.1016/j.cjca.2019.10.037. Epub 2019 Nov 4.
Abstract
摘要
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本文引用的文献
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Can J Cardiol. 2020 Jun;36(6):893-905. doi: 10.1016/j.cjca.2019.09.026. Epub 2019 Nov 1.
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Inflammation following acute myocardial infarction: Multiple players, dynamic roles, and novel therapeutic opportunities.急性心肌梗死后的炎症反应：多种角色，动态作用，以及新的治疗机会。
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The NLRP3 inflammasome in acute myocardial infarction.NLRP3 炎性小体与急性心肌梗死。
Nat Rev Cardiol. 2018 Apr;15(4):203-214. doi: 10.1038/nrcardio.2017.161. Epub 2017 Nov 16.
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Inflammation as a Driver of Adverse Left Ventricular Remodeling After Acute Myocardial Infarction.炎症作为急性心肌梗死后不良左心室重构的驱动因素。
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