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Two alleles of locus disrupting potential cell-binding motif of UNC-52.

作者信息

Wilsey Rachel, Hodge Sabrina, Kenney Krysta, Wahl Jacob, Jaffery Roshni, Brau Avery, Qiu Zhongqiang, Lee Myeongwoo

机构信息

One Bear Place 97388, Department of Biology, Baylor University, Waco, TX 76798, U.S.A.

出版信息

MicroPubl Biol. 2020 May 17;2020. doi: 10.17912/micropub.biology.000250.

DOI:10.17912/micropub.biology.000250
PMID:32550495
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7252330/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c1f5/7252330/bfaf41700c4d/25789430-2020-micropub.biology.000250.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c1f5/7252330/bfaf41700c4d/25789430-2020-micropub.biology.000250.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/c1f5/7252330/bfaf41700c4d/25789430-2020-micropub.biology.000250.jpg

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1
Two alleles of locus disrupting potential cell-binding motif of UNC-52.UNC-52位点的两个等位基因破坏了潜在的细胞结合基序。
MicroPubl Biol. 2020 May 17;2020. doi: 10.17912/micropub.biology.000250.
2
Behavioral genetics of caenorhabditis elegans unc-103-encoded erg-like K(+) channel.秀丽隐杆线虫unc-103编码的类Erg钾通道的行为遗传学
J Neurogenet. 2006 Jan-Jun;20(1-2):41-66. doi: 10.1080/01677060600788826.
3
UNC-5 function requires phosphorylation of cytoplasmic tyrosine 482, but its UNC-40-independent functions also require a region between the ZU-5 and death domains.UNC-5的功能需要胞质酪氨酸482的磷酸化，但其不依赖UNC-40的功能也需要位于ZU-5结构域和死亡结构域之间的一个区域。
Dev Biol. 2002 Nov 15;251(2):348-66. doi: 10.1006/dbio.2002.0825.
4
The unc-18 gene encodes a novel protein affecting the kinetics of acetylcholine metabolism in the nematode Caenorhabditis elegans.unc-18基因编码一种影响线虫秀丽隐杆线虫乙酰胆碱代谢动力学的新型蛋白质。
J Neurochem. 1992 Apr;58(4):1517-25. doi: 10.1111/j.1471-4159.1992.tb11373.x.
5
UNC-52/perlecan affects gonadal leader cell migrations in C. elegans hermaphrodites through alterations in growth factor signaling.UNC-52/基底膜聚糖通过改变生长因子信号传导影响秀丽隐杆线虫雌雄同体性腺引导细胞的迁移。
Dev Biol. 2003 Apr 1;256(1):173-86. doi: 10.1016/s0012-1606(03)00014-9.
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Identification of domains of netrin UNC-6 that mediate attractive and repulsive guidance and responses from cells and growth cones.鉴定介导吸引和排斥性导向以及细胞和生长锥反应的UNC-6网蛋白结构域。
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Critical residues of the Caenorhabditis elegans unc-2 voltage-gated calcium channel that affect behavioral and physiological properties.影响秀丽隐杆线虫行为和生理特性的unc-2电压门控钙通道的关键残基。
J Neurosci. 2003 Jul 23;23(16):6537-45. doi: 10.1523/JNEUROSCI.23-16-06537.2003.
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The netrin receptor UNC-40/DCC stimulates axon attraction and outgrowth through enabled and, in parallel, Rac and UNC-115/AbLIM.神经营养因子受体UNC-40/DCC通过Enabled以及平行的Rac和UNC-115/AbLIM刺激轴突吸引和生长。
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Genetic analysis of halothane sensitivity in Caenorhabditis elegans.秀丽隐杆线虫中氟烷敏感性的遗传分析。
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本文引用的文献

1
Modular Proteoglycan Perlecan/: Mutations, Phenotypes, and Functions.模块化蛋白聚糖基底膜聚糖：突变、表型与功能
Genes (Basel). 2018 Nov 16;9(11):556. doi: 10.3390/genes9110556.
2
High Efficiency, Homology-Directed Genome Editing in Caenorhabditis elegans Using CRISPR-Cas9 Ribonucleoprotein Complexes.利用CRISPR-Cas9核糖核蛋白复合物在秀丽隐杆线虫中进行高效、同源定向基因组编辑
Genetics. 2015 Sep;201(1):47-54. doi: 10.1534/genetics.115.179382. Epub 2015 Jul 17.
3
Efficient marker-free recovery of custom genetic modifications with CRISPR/Cas9 in Caenorhabditis elegans.
利用CRISPR/Cas9在秀丽隐杆线虫中高效无标记回收定制基因修饰。
Genetics. 2014 Nov;198(3):837-46. doi: 10.1534/genetics.114.169730. Epub 2014 Aug 26.
4
A novel mutation in β integrin reveals an integrin-mediated interaction between the extracellular matrix and cki-1/p27KIP1.一种新型的β整合素突变揭示了细胞外基质和 cki-1/p27KIP1 之间的整合素介导的相互作用。
PLoS One. 2012;7(8):e42425. doi: 10.1371/journal.pone.0042425. Epub 2012 Aug 6.
5
The RGD motif in fibronectin is essential for development but dispensable for fibril assembly.纤连蛋白中的RGD基序对发育至关重要，但对纤维组装并非必需。
J Cell Biol. 2007 Jul 2;178(1):167-78. doi: 10.1083/jcb.200703021. Epub 2007 Jun 25.
6
Spectrum of HSPG2 (Perlecan) mutations in patients with Schwartz-Jampel syndrome.施瓦茨-扬佩尔综合征患者中HSPG2（基底膜聚糖）突变谱。
Hum Mutat. 2006 Nov;27(11):1082-91. doi: 10.1002/humu.20388.
7
Dyssegmental dysplasia, Silverman-Handmaker type, is caused by functional null mutations of the perlecan gene.节段性发育异常，Silverman-Handmaker型，由基底膜聚糖基因的功能缺失性突变引起。
Nat Genet. 2001 Apr;27(4):431-4. doi: 10.1038/86941.
8
Complex patterns of alternative splicing mediate the spatial and temporal distribution of perlecan/UNC-52 in Caenorhabditis elegans.复杂的可变剪接模式介导了秀丽隐杆线虫中基底膜聚糖/UNC-52的时空分布。
Mol Biol Cell. 1999 Oct;10(10):3205-21. doi: 10.1091/mbc.10.10.3205.
9
Products of the unc-52 gene in Caenorhabditis elegans are homologous to the core protein of the mammalian basement membrane heparan sulfate proteoglycan.秀丽隐杆线虫中unc-52基因的产物与哺乳动物基底膜硫酸乙酰肝素蛋白聚糖的核心蛋白同源。
Genes Dev. 1993 Aug;7(8):1471-84. doi: 10.1101/gad.7.8.1471.
10
Mutations in the unc-52 gene responsible for body wall muscle defects in adult Caenorhabditis elegans are located in alternatively spliced exons.导致成年秀丽隐杆线虫体壁肌肉缺陷的unc-52基因中的突变位于可变剪接外显子中。
Genetics. 1995 Jan;139(1):159-69. doi: 10.1093/genetics/139.1.159.