Effects of islet-activating protein on the catecholamine release, Ca2+ mobilization and inositol trisphosphate formation in cultured adrenal chromaffin cells.

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作者信息

Sasakawa N, Yamamoto S, Nakaki T, Kato R

机构信息

Department of Pharmacology, School of Medicine, Keio University, Tokyo, Japan.

出版信息

Biochem Pharmacol. 1988 Jun 15;37(12):2485-7. doi: 10.1016/0006-2952(88)90379-6.

DOI:10.1016/0006-2952(88)90379-6

PMID:3260497

Abstract

摘要

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Effects of islet-activating protein on the catecholamine release, Ca2+ mobilization and inositol trisphosphate formation in cultured adrenal chromaffin cells.

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Inositol trisphosphate mobilizes intracellular calcium in permeabilized adrenal chromaffin cells.肌醇三磷酸在透化的肾上腺嗜铬细胞中动员细胞内钙。

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Cholinergic stimulation of inositol phosphate formation in bovine adrenal chromaffin cells: distinct nicotinic and muscarinic mechanisms.牛肾上腺嗜铬细胞中胆碱能刺激肌醇磷酸形成：不同的烟碱样和毒蕈碱样机制

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Stimulation of formation of inositol phosphates in primary cultures of bovine adrenal chromaffin cells by angiotensin II, histamine, bradykinin, and carbachol.血管紧张素II、组胺、缓激肽和卡巴胆碱对牛肾上腺嗜铬细胞原代培养物中肌醇磷酸形成的刺激作用。

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Islet activating protein inhibits kinin-stimulated inositol phosphate production, calcium mobilization, and prostaglandin E2 synthesis in renal papillary collecting tubule cells independent of cyclic AMP.胰岛激活蛋白可抑制肾乳头集合管细胞中缓激肽刺激的肌醇磷酸生成、钙动员及前列腺素E2合成，且不依赖于环磷酸腺苷。

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Calcium-independent desensitization of rises in intracellular free Ca2+ concentration and catecholamine release in cultured adrenal chromaffin cells.

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Biochem J. 1991 Mar 1;274 ( Pt 2)(Pt 2):339-47. doi: 10.1042/bj2740339.

Pertussis toxin stimulation of catecholamine release from adrenal medullary chromaffin cells: mechanism may be by direct activation of L-type and G-type calcium channels.百日咳毒素刺激肾上腺髓质嗜铬细胞释放儿茶酚胺：其机制可能是直接激活L型和G型钙通道。

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