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Sera From Patients With Minimal Change Disease Increase Endothelial Permeability to Sodium.

作者信息

Daviet Florence, Blin Muriel G, Fallague Karim, Bachelier Richard, Laforêt Manon, Carré Manon, Poitevin Stéphane, Dignat-George Françoise, Blot-Chabaud Marcel, Bardin Nathalie, Burtey Stéphane, Jourde-Chiche Noémie, Leroyer Aurélie S

机构信息

Aix-Marseille University, Centre de recherche en CardioVasculaire et Nutrition, Institut national de la santé et de la recherche médicale 1263, Institut national de recherche pour l'agriculture, l'alimentation et l'environnement 1260, Marseille, France.

Assistance Publique-Hôpitaux de Marseille, Centre de Néphrologie et Transplantation Rénale, Hôpital de la Conception, Marseille, France.

出版信息

Kidney Int Rep. 2020 Apr 20;5(7):1071-1075. doi: 10.1016/j.ekir.2020.04.010. eCollection 2020 Jul.

DOI:10.1016/j.ekir.2020.04.010
PMID:32647766
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7335967/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6408/7335967/ce5e2e8e0947/gr3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6408/7335967/717e8e08c123/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6408/7335967/3046b4c8fbe6/gr2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6408/7335967/ce5e2e8e0947/gr3.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6408/7335967/717e8e08c123/gr1.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6408/7335967/3046b4c8fbe6/gr2.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/6408/7335967/ce5e2e8e0947/gr3.jpg

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引用本文的文献

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本文引用的文献

1
Proteasuria-The impact of active urinary proteases on sodium retention in nephrotic syndrome.蛋白尿——尿中活性蛋白酶对肾病综合征钠潴留的影响。
Acta Physiol (Oxf). 2019 Apr;225(4):e13249. doi: 10.1111/apha.13249. Epub 2019 Jan 18.
2
Aprotinin prevents proteolytic epithelial sodium channel (ENaC) activation and volume retention in nephrotic syndrome.抑肽酶可预防肾病综合征中蛋白水解上皮钠离子通道（ENaC）的激活和容量潴留。
Kidney Int. 2018 Jan;93(1):159-172. doi: 10.1016/j.kint.2017.07.023. Epub 2017 Oct 14.
3
Pathophysiology, Evaluation, and Management of Edema in Childhood Nephrotic Syndrome.
儿童肾病综合征水肿的病理生理学、评估和管理。
Front Pediatr. 2016 Jan 11;3:111. doi: 10.3389/fped.2015.00111. eCollection 2015.
4
Permeability factors in idiopathic nephrotic syndrome: historical perspectives and lessons for the future.特发性肾病综合征的通透性因素：历史观点和对未来的启示。
Nephrol Dial Transplant. 2014 Dec;29(12):2207-16. doi: 10.1093/ndt/gfu355.
5
Blood pressure and amiloride-sensitive sodium channels in vascular and renal cells.血管和肾细胞中的血压与阿米洛利敏感的钠通道
Nat Rev Nephrol. 2014 Mar;10(3):146-57. doi: 10.1038/nrneph.2013.275. Epub 2014 Jan 14.
6
Cellular and molecular regulation of vascular permeability.血管通透性的细胞和分子调节。
Thromb Haemost. 2013 Mar;109(3):407-15. doi: 10.1160/TH12-09-0678. Epub 2013 Feb 7.
7
Caveolae, caveolins, cavins, and endothelial cell function: new insights.小窝、小窝蛋白、窖蛋白及内皮细胞功能：新见解
Front Physiol. 2012 Jan 6;2:120. doi: 10.3389/fphys.2011.00120. eCollection 2012.
8
Plasmin in nephrotic urine activates the epithelial sodium channel.肾病尿液中的纤溶酶激活上皮钠通道。
J Am Soc Nephrol. 2009 Feb;20(2):299-310. doi: 10.1681/ASN.2008040364. Epub 2008 Dec 10.
9
Hyperaldosteronemia and activation of the epithelial sodium channel are not required for sodium retention in puromycin-induced nephrosis.在嘌呤霉素诱导的肾病中，钠潴留并不需要高醛固酮血症和上皮钠通道的激活。
J Am Soc Nephrol. 2005 Dec;16(12):3642-50. doi: 10.1681/ASN.2005040363. Epub 2005 Nov 2.