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抑制巨噬细胞移动抑制因子可预防急性胰腺炎孕鼠甲状腺功能障碍。

Inhibition of macrophage migration inhibitory factor prevents thyroid dysfunction in pregnant rats with acute pancreatitis.

机构信息

Department of General Surgery, Renmin Hospital of Wuhan University, Wuhan 430060, Hubei, China.

Department of General Surgery, Renmin Hospital of Wuhan University, Wuhan 430060, Hubei, China; Key Laboratory of Hubei Province for Digestive System Disease, Wuhan 430060, Hubei, China.

出版信息

Int Immunopharmacol. 2020 Oct;87:106771. doi: 10.1016/j.intimp.2020.106771. Epub 2020 Jul 16.

DOI:10.1016/j.intimp.2020.106771
PMID:32683302
Abstract

Acute pancreatitis during pregnancy (APIP) rarely occurs but may lead to preterm delivery and be associated with high fetal mortality. Macrophage migration inhibitory factor (MIF) participates in various inflammatory diseases as a pro-inflammatory cytokine. In this study, we aimed to explore the effects of (S, R)-3-(4-hydroxyphenyl)-4, 5dihydro-5-isoxazole acetic methyl ester (ISO-1), an inhibitor of MIF, on maternal thyroid injury associated with APIP and its potential mechanisms in a pregnant rat model. APIP model was induced by retrograde injection of sodium taurocholate. ISO-1 was injected intraperitoneally 30 min before model establishment. The severity of pancreatitis was assessed by levels of tumor necrosis factor (TNF)‑α, interleukin (IL)‑1β, IL-6 of maternal serum as well as histopathological score. Thyroid injury was determined by free triiodothyronine (FT3), free tetraiodothyronine (FT4) and thyroid histopathological score. Levels of MIF in maternal serum and the expression of MIF, CD68, CD3 and intercellular cell adhesion molecule-1 (ICAM-1) as well as oxidative stress status in maternal thyroid tissues were detected. Ultrastructure of maternal thyroid tissues was observed by transmission electron microscope. Thyroid injuries occurred in APIP and the lesions were attenuated with the pretreatment of ISO-1. Moreover, ISO-1 reduced the expression of MIF, attenuated the activations of CD68, CD3, ICAM-1 while improved oxidative stress status in maternal thyroid. Our research suggested a protective role of ISO-1 on thyroid injury and endocrine disorder during APIP, which may be associated with the inhibition of biological functions of MIF.

摘要

妊娠期急性胰腺炎(APIP)很少见,但可能导致早产,并与胎儿死亡率高有关。巨噬细胞移动抑制因子(MIF)作为一种促炎细胞因子,参与多种炎症性疾病。在这项研究中,我们旨在探讨(S,R)-3-(4-羟基苯基)-4,5-二氢-5-异恶唑乙酸甲酯(ISO-1),一种 MIF 抑制剂,对伴有 APIP 的母体甲状腺损伤及其在妊娠大鼠模型中的潜在机制的影响。APIP 模型通过逆行注射牛磺胆酸钠诱导。ISO-1 在模型建立前 30 分钟腹腔注射。通过检测母血清中肿瘤坏死因子(TNF)-α、白细胞介素(IL)-1β、IL-6 的水平以及组织病理学评分来评估胰腺炎的严重程度。通过游离三碘甲状腺原氨酸(FT3)、游离甲状腺素(FT4)和甲状腺组织病理学评分来确定甲状腺损伤。检测母血清中 MIF 水平以及 MIF、CD68、CD3 和细胞间黏附分子-1(ICAM-1)的表达和母甲状腺组织的氧化应激状态。通过透射电镜观察母甲状腺组织的超微结构。APIP 发生甲状腺损伤,ISO-1 预处理可减轻损伤。此外,ISO-1 降低了 MIF 的表达,减弱了 CD68、CD3、ICAM-1 的激活,同时改善了母甲状腺的氧化应激状态。我们的研究表明,ISO-1 在 APIP 期间对甲状腺损伤和内分泌紊乱具有保护作用,这可能与抑制 MIF 的生物学功能有关。

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