Wakuta Naoki, Yamamoto Satoshi
Department of Neurosurgery, Okinawa Miyako Hospital, Okinawa, Japan.
Department of Neurosurgery, Fukuoka University Hospital, School of Medicine, Fukuoka University, Fukuoka, Japan.
SAGE Open Med Case Rep. 2020 Jul 15;8:2050313X20933752. doi: 10.1177/2050313X20933752. eCollection 2020.
A 65-year-old male received coil embolization for a large internal carotid-posterior communicating artery aneurysm. Pre- and postoperative angiography at surgery demonstrated that the ipsilateral anterior choroidal artery branched from the internal carotid artery near the distal side of the aneurysm, and elevated and expanded on the aneurysmal dome, but was clearly visualized. Three days following endovascular treatment, the patient presented hemiparesis on the left side, with brain infarction in the territory of the right anterior choroidal artery despite antithrombotic therapy. The delayed brain infarction was likely caused by a reduction in anterior choroidal artery perfusion caused by mechanical compression following a postoperative increase in internal carotid-posterior communicating artery aneurysmal volume during intra-aneurysmal thrombosis. Transient volume expansion after coil embolization for intracranial aneurysms is rarely reported as a cause of brain infarction. It is important to recognize these arteries as potential postoperative complication risks, and consider the use of open surgery to avoid this risk.
一名65岁男性因大型颈内动脉-后交通动脉瘤接受了弹簧圈栓塞治疗。手术前后的血管造影显示,同侧脉络膜前动脉在动脉瘤远端附近从颈内动脉分支,并在动脉瘤瘤顶上抬高和扩张,但仍清晰可见。血管内治疗三天后,尽管进行了抗血栓治疗,患者仍出现左侧偏瘫,右侧脉络膜前动脉供血区发生脑梗死。延迟性脑梗死可能是由于动脉瘤内血栓形成过程中,颈内动脉-后交通动脉瘤术后体积增加导致机械压迫,引起脉络膜前动脉灌注减少所致。颅内动脉瘤弹簧圈栓塞术后短暂的体积扩张作为脑梗死的原因鲜有报道。认识到这些动脉作为术后潜在并发症风险很重要,并考虑采用开放手术以避免这种风险。