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Cigarette smoking, lung inflammation, and the development of emphysema.

作者信息

Niewoehner D E

机构信息

Pulmonary Section, Veterans Administration Medical Center, Minneapolis, MN 55417.

出版信息

J Lab Clin Med. 1988 Jan;111(1):15-27.

PMID:3275730
Abstract
摘要

相似文献

1
Cigarette smoking, lung inflammation, and the development of emphysema.
J Lab Clin Med. 1988 Jan;111(1):15-27.
2
[Smokers with chronic bronchitis and emphysema. Utilize the entire therapy spectrum!].[患有慢性支气管炎和肺气肿的吸烟者。采用全面的治疗方案!]
MMW Fortschr Med. 2000 Feb 10;142(6):30-3.
3
[Pulmonary emphysema and obstructive respiratory tract disease: etiology--diagnosis--prevention].
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[Smoking cessation in unavoidable. Interview by Dr. rer. nat. Renate Leinmüller].[戒烟势在必行。自然科学博士蕾娜特·莱因米勒访谈]
Fortschr Med. 1998 May 20;116(14):28-30.
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The effect of smoking cessation and steroid treatment on emphysema in guinea pigs.戒烟和类固醇治疗对豚鼠肺气肿的影响。
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6
[Does the manner of smoking affect chronic obstructive airway diseases and bronchial cancer?].[吸烟方式是否会影响慢性阻塞性气道疾病和支气管癌?]
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7
Pathophysiology of obstructive airways disease.
Radiol Clin North Am. 1998 Jan;36(1):15-27. doi: 10.1016/s0033-8389(05)70005-1.
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Factors influencing airway inflammation in chronic obstructive pulmonary disease.影响慢性阻塞性肺疾病气道炎症的因素
Thorax. 2000 Nov;55(11):970-7. doi: 10.1136/thorax.55.11.970.
9
[Lung tissue changes in irreversible airflow obstruction--emphysema and small airway disease].[不可逆性气流受限中的肺组织改变——肺气肿和小气道疾病]
Duodecim. 1993;109(12):1061-9.
10
Increased surfactant protein-D and foamy macrophages in smoking-induced mouse emphysema.吸烟诱导的小鼠肺气肿中表面活性蛋白-D和泡沫巨噬细胞增加。
Respirology. 2007 Mar;12(2):191-201. doi: 10.1111/j.1440-1843.2006.01009.x.

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Are lung disease and function related to age-related macular degeneration?肺部疾病和功能与年龄相关性黄斑变性有关吗?
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最新技术。通过多排探测器计算机断层扫描对肺部进行结构和功能评估:慢性阻塞性肺疾病的表型分析。
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Cellular events in alveolitis and the evolution of pulmonary fibrosis.肺泡炎中的细胞事件与肺纤维化的演变
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Detection of lipocortin 1 in human lung lavage fluid: lipocortin degradation as a possible proteolytic mechanism in the control of inflammatory mediators and inflammation.
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Marked longevity of human lung parenchymal elastic fibers deduced from prevalence of D-aspartate and nuclear weapons-related radiocarbon.从D-天冬氨酸的普遍存在及与核武器相关的放射性碳推断出人类肺实质弹性纤维显著的长寿特性。
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