State Key Laboratory for Biology of Plant Diseases and Insect Pests, Institute of Plant Protection, Chinese Academy of Agricultural Sciences, Beijing 100193, China; Department of Plant Pathology, the Ohio State University, Columbus, OH 43210, USA.
State Key Laboratory for Biology of Plant Diseases and Insect Pests, Institute of Plant Protection, Chinese Academy of Agricultural Sciences, Beijing 100193, China.
Trends Plant Sci. 2020 Nov;25(11):1060-1062. doi: 10.1016/j.tplants.2020.08.001. Epub 2020 Aug 26.
Activation of plant respiratory burst oxidase homologs (RBOHs) to generate reactive oxygen species (ROS) is a crucial defense signaling event. RBOH activation occurs predominantly through N-terminal phosphorylation and the binding of a small GTPase. Two recent papers reported that C-terminal phosphorylation and ubiquitination modulates AtRBOHD activity, which extends our understanding of the fine-tuning of RBOH signaling in plant immunity.
植物呼吸爆发氧化酶同源物（RBOHs）的激活以产生活性氧物种（ROS）是一个关键的防御信号事件。RBOH 的激活主要通过 N 端磷酸化和小 GTP 结合发生。最近的两篇论文报道 C 端磷酸化和泛素化调节 AtRBOHD 的活性，这扩展了我们对植物免疫中 RBOH 信号精细调控的理解。
