Peters Futre E M, Noakes T D, Raine R I, Terblanche S E
Am J Physiol. 1987 Sep;253(3 Pt 1):E305-11. doi: 10.1152/ajpendo.1987.253.3.E305.
High-intensity intermittent bicycle exercise was used to deplete muscle glycogen levels by 70% and elevate blood lactate levels to greater than 13.0 mmol/l. Thereafter subjects either cycled with one leg for 45 min followed by 45 min of passive recovery (partially active recovery) or rested for 90 min (passive recovery). During the first 45 min of partially active recovery 1) blood lactate (P less than 0.05) and pH levels (P less than 0.05) returned more rapidly to preexercise values than during passive recovery, 2) the rate of net glycogen resynthesis (0.28 mumol . g-1 . min-1) was the same in both legs, and 3) muscle lactate levels were significantly lower (P less than 0.05) in the passive than in the active leg. Thereafter the rate of net muscle glycogen resynthesis was unchanged (0.26 mumol . g-1 . min-1) and lactate removal could theoretically account for only 18% of the glycogen resynthesized. Overall, the rate of muscle glycogen resynthesis and muscle lactate removal was not different from that measured during passive recovery. After high-intensity exercise 1) glycogen repletion is not impeded by light exercise, and 2) blood glucose is an important substrate for glycogen resynthesis.
采用高强度间歇自行车运动使肌肉糖原水平降低70%，并将血乳酸水平升高至大于13.0 mmol/L。此后，受试者要么单腿骑行45分钟，然后进行45分钟的被动恢复（部分主动恢复），要么休息90分钟（被动恢复）。在部分主动恢复的前45分钟内，1）血乳酸（P<0.05）和pH值水平（P<0.05）比被动恢复时更快地恢复到运动前值，2）双腿净糖原再合成速率（0.28 μmol·g-1·min-1）相同，3）被动腿的肌肉乳酸水平显著低于主动腿（P<0.05）。此后，净肌肉糖原再合成速率保持不变（0.26 μmol·g-1·min-1），理论上乳酸清除仅占再合成糖原的18%。总体而言，肌肉糖原再合成和肌肉乳酸清除率与被动恢复期间测得的速率没有差异。高强度运动后，1）轻度运动不会阻碍糖原补充，2）血糖是糖原再合成的重要底物。
