Raghavendra B N, Young B K, Greco M A, Lustig-Gillman I, Horii S C, Hirsch M A, Yee J
Department of Radiology, New York University School of Medicine, NY 10016.
Radiology. 1987 Nov;165(2):455-8. doi: 10.1148/radiology.165.2.3310099.
To differentiate pregnancies complicated by oligohydramnios due to intrauterine growth retardation from those due to renal agenesis, the authors administered furosemide intravenously to eight pregnant women (19-25 weeks gestation) with oligohydramnios. The fetal abdomen was scanned with ultrasound to demonstrate the fetal urinary bladder. In six fetuses, sonography failed to demonstrate the bladder: two fetuses had growth retardation with normal kidneys and bladder, and four had renal anomalies. It is concluded that administration of furosemide to the mother fails to induce diuresis in growth-retarded fetuses of 19-23 weeks gestation and that failure to see the fetal bladder after furosemide administration does not necessarily indicate absent fetal kidneys.
为了鉴别因宫内生长迟缓导致羊水过少的妊娠与因肾缺如导致羊水过少的妊娠，作者对8例羊水过少的孕妇（妊娠19 - 25周）静脉注射速尿。用超声扫描胎儿腹部以显示胎儿膀胱。在6例胎儿中，超声检查未能显示膀胱：2例胎儿生长迟缓但肾脏和膀胱正常，4例有肾脏异常。得出的结论是，对母亲注射速尿未能在妊娠19 - 23周生长迟缓的胎儿中诱导利尿，且注射速尿后未见到胎儿膀胱不一定表明胎儿肾脏缺如。
