Takano S, Nose T, Maki Y, Shinohara A, Kukita C
Department of Neurosurgery, University of Tukuba, Ibaraki, Japan.
No Shinkei Geka. 1987 Sep;15(9):991-6.
The authors report a case of spontaneous occlusion of an arteriovenous malformation (AVM) verified by the second angiography performed 3 days after the initial one. This 65-year-old man had a sudden attack of headache, nausea, and vomiting and was admitted to our hospital next day. On admission, CT scan showed subcortical hemorrhage in the right temporo-parietal area and right CAG showed a small AVM in the same area. The main feeder was a MCA distal branch and the drainer joined Labbe's vein. Repeated angiography 3 days after initial one failed to demonstrate the AVM. Craniotomy was performed and thrombosed AVM was totally removed. Mechanism for disappearance of the malformation is assumed to be acute thrombosis due to intracranial hemorrhage and arteriosclerotic change. The literature is reviewed.
作者报告了一例动静脉畸形（AVM）自发闭塞的病例，该病例通过首次血管造影3天后进行的第二次血管造影得以证实。这位65岁的男性突然出现头痛、恶心和呕吐，次日入院。入院时，CT扫描显示右侧颞顶叶区域皮质下出血，右侧脑血管造影（CAG）显示同一区域有一个小的AVM。主要供血支为大脑中动脉（MCA）远端分支，引流静脉汇入Labbe静脉。首次血管造影3天后重复血管造影未显示AVM。遂行开颅手术，将血栓形成的AVM完全切除。畸形消失的机制推测为颅内出血和动脉硬化改变导致的急性血栓形成。并对相关文献进行了综述。
