Carr Peter
Specialty Psychiatrist & Medical Psychotherapist, Psychotherapy & Eating Disorder Services, Greenfields House, New Craigs Hospital, 6-16 Leachkin Road, Inverness IV3 8NP, United Kingdom.
Med Hypotheses. 2020 Nov;144:110184. doi: 10.1016/j.mehy.2020.110184. Epub 2020 Aug 16.
The value of addressing trauma-related issues in psychosis with therapies such as ṇEMDR is being increasingly recognised. The hypothesis of this paper is that augmentation of EMDR with alternating bilateral photic stimulation (ABPS) is especially suited to those on the schizotypal spectrum. Not only does Alternating Bilateral Photic Stimulation (ABPS) at alpha frequencies (8-12 Hz) directly compensate EEG deficits of schizophrenia by photic driving, it also stimulates pseudo-hallucinatory visual imagery (visions) and a waking dream state. In contrast to the REM-sleep state, the dorsolateral prefrontal cortex remains activated during the flicker-induced waking dream and its functions of reflective thought and insight are preserved. Recent EEG and neuroimaging studies are providing empirical support for the long-held conjecture that psychosis is caused by a conflation of REM-sleep / dreaming and waking states. Llewellyn's theory of de-differentiation between sleep-dreaming and waking accounts for both the creativity and psychopathology of the schizotypy spectrum. It is hypothesised that flicker-induced hypnagogic imagery reinforces the EMDR mechanism of action proposed by Stickgold, namely facilitation of memory integration during sleep, by creating greater linkage between EMDR therapy and dreaming. With ABPS-induced visioning EMDR progresses seamlessly from initial targeting of trauma and adversity to desensitisation of emotionally-charged brain-disturbing material related to identity, selfhood, and embodiment. Desensitisation of the affect allows the self-regulatory dynamics of sleep-dreaming to restore embodied selfhood by an integrative process of resynchronisation. This in turn leads to re-differentiation of waking embodiment from dream-embodiment. Evolutionary theories as to the aetiology of the schizotypy spectrum are positing a genetic origin in indigenous shamanic religions which are based on visioning ability. Patients with schizophrenia score better than normal controls on vividness of visual imagery and ability to generate, inspect, and manipulate mental images. Self-help EMDR glasses can be provided to patients for long-term regular practice of ABPS-induced visioning. It is proposed that many on the schizotypal spectrum, including some at the severe end, can improve and maintain their mental health by the regular practice of ABPS-induced visioning, and thereby transition progressively over time from negative to positive schizotypy.
人们越来越认识到,使用诸如眼动脱敏再处理疗法(ṇEMDR)等疗法来解决精神病中与创伤相关问题的价值。本文的假设是,用交替双侧光刺激(ABPS)增强眼动脱敏再处理疗法特别适合分裂型谱系的患者。不仅在阿尔法频率(8 - 12赫兹)下的交替双侧光刺激(ABPS)通过光驱动直接补偿精神分裂症的脑电图缺陷,它还能刺激假性幻觉视觉意象(幻象)和清醒梦状态。与快速眼动睡眠状态不同,背外侧前额叶皮层在闪烁诱导的清醒梦中保持激活,其反思性思维和洞察力功能得以保留。最近的脑电图和神经影像学研究为长期以来的推测提供了实证支持,即精神病是由快速眼动睡眠/做梦和清醒状态的混合导致的。卢埃林关于睡眠 - 做梦和清醒之间去分化的理论解释了分裂型谱系的创造力和精神病理学。据推测,闪烁诱导的入睡幻觉意象通过在眼动脱敏再处理疗法和做梦之间建立更大的联系,强化了斯蒂克戈尔德提出的眼动脱敏再处理疗法的作用机制,即在睡眠期间促进记忆整合。通过ABPS诱导的视觉化,眼动脱敏再处理疗法从最初针对创伤和逆境无缝进展到对与身份、自我和体现相关的情绪激动的大脑干扰物质进行脱敏。情感的脱敏使睡眠 - 做梦的自我调节动态通过重新同步的整合过程恢复体现的自我。这反过来又导致清醒体现与梦境体现的重新分化。关于分裂型谱系病因的进化理论假定其在基于视觉化能力的本土萨满教中有遗传起源。精神分裂症患者在视觉意象的生动性以及生成、检查和操纵心理意象的能力方面得分高于正常对照组。可以为患者提供自助眼动脱敏再处理疗法眼镜,以便长期定期进行ABPS诱导的视觉化练习。有人提出,许多分裂型谱系的患者,包括一些病情严重的患者,可以通过定期进行ABPS诱导的视觉化练习来改善和维持他们的心理健康,从而随着时间的推移逐渐从消极的分裂型转变为积极的分裂型。