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铁与藤茶中提取的二氢杨梅素之间的相互作用。

Interaction between iron and dihydromyricetin extracted from vine tea.

作者信息

Wang Liling, Qin Yuchuan, Wang Yanbin, Zhou Yifeng, Liu Bentong

机构信息

Zhejiang Academy of Forestry Hangzhou China.

School of Biological and Chemical Engineering Zhejiang University of Science and Technology Hangzhou China.

出版信息

Food Sci Nutr. 2020 Sep 13;8(11):5926-5933. doi: 10.1002/fsn3.1876. eCollection 2020 Nov.


DOI:10.1002/fsn3.1876
PMID:33282244
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7684613/
Abstract

In this research, the interaction between dihydromyricetin (DMY) obtained from vine tea and iron ions (Fe (II) and Fe (III)) was investigated at pH 3.0, 5.0, and 7.0 with UV absorption and fluorescence quenching spectroscopy. The effects of DMY on the stability and solubility of iron ion were also studied. The results showed the presence of iron ions changed the UV absorption spectra of DMY at the experimental pH values. And the fluorescence spectra showed that iron ion had enhanced fluorescence effect on DMY. In addition, DMY was capable of protecting Fe (II) from being oxidized and improving the solubility of Fe (III).

摘要

本研究利用紫外吸收光谱和荧光猝灭光谱,在pH值为3.0、5.0和7.0的条件下,研究了从藤茶中提取的二氢杨梅素(DMY)与铁离子(Fe (II) 和Fe (III))之间的相互作用。同时还研究了DMY对铁离子稳定性和溶解性的影响。结果表明,在实验pH值条件下,铁离子的存在改变了DMY的紫外吸收光谱。荧光光谱显示,铁离子对DMY有增强荧光的作用。此外,DMY能够保护Fe (II) 不被氧化,并提高Fe (III) 的溶解性。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0f9a/7684613/fd485bad0920/FSN3-8-5926-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0f9a/7684613/8517d19547c4/FSN3-8-5926-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0f9a/7684613/1cdecb382fda/FSN3-8-5926-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0f9a/7684613/6d3c24d9ba41/FSN3-8-5926-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0f9a/7684613/4a5522b23b9e/FSN3-8-5926-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0f9a/7684613/fd485bad0920/FSN3-8-5926-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0f9a/7684613/8517d19547c4/FSN3-8-5926-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0f9a/7684613/1cdecb382fda/FSN3-8-5926-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0f9a/7684613/6d3c24d9ba41/FSN3-8-5926-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0f9a/7684613/4a5522b23b9e/FSN3-8-5926-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/0f9a/7684613/fd485bad0920/FSN3-8-5926-g005.jpg

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[1]
Interaction between iron and dihydromyricetin extracted from vine tea.

Food Sci Nutr. 2020-9-13

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引用本文的文献

[1]
Influence of the Maillard Reaction on the Properties of Gelatin/Zein Nanofibers Loaded with Dihydromyricetin Prepared by Electro-Blowing Spinning.

Biomolecules. 2025-6-18

[2]
LC-ESI-QTOF/MS characterization of antimicrobial compounds with their action mode extracted from vine tea () leaves.

Food Sci Nutr. 2022-1-19

[3]
Spectroscopic and in silico investigation of the interaction between GH1 β-glucosidase and ginsenoside Rb.

Food Sci Nutr. 2021-2-9

本文引用的文献

[1]
Dihydromyricetin Attenuates Metabolic Syndrome And Improves Insulin Sensitivity By Upregulating Insulin Receptor Substrate-1 (Y612) Tyrosine Phosphorylation In db/db Mice.

Diabetes Metab Syndr Obes. 2019-11-1

[2]
Golgi reassembly and stacking protein 65 downregulation is required for the anti-cancer effect of dihydromyricetin on human ovarian cancer cells.

PLoS One. 2019-11-26

[3]
Dihydromyricetin Alleviates Diabetic Neuropathic Pain and Depression Comorbidity Symptoms by Inhibiting P2X Receptor.

Front Psychiatry. 2019-10-18

[4]
Coordination of Fe to Eugenol to Engineer Self-Assembled Emulsions by Rice Proteins for Iron Fortification.

J Food Sci. 2019-1-16

[5]
Protective role of Dihydromyricetin in Alzheimer's disease rat model associated with activating AMPK/SIRT1 signaling pathway.

Biosci Rep. 2019-1-3

[6]
Dihydromyricetin prevents monocrotaline-induced pulmonary arterial hypertension in rats.

Biomed Pharmacother. 2017-11-6

[7]
Metabolomics reveals that vine tea (Ampelopsis grossedentata) prevents high-fat-diet-induced metabolism disorder by improving glucose homeostasis in rats.

PLoS One. 2017-8-16

[8]
Targeting P-glycoprotein and SORCIN: Dihydromyricetin strengthens anti-proliferative efficiency of adriamycin via MAPK/ERK and Ca -mediated apoptosis pathways in MCF-7/ADR and K562/ADR.

J Cell Physiol. 2017-11-14

[9]
Dihydromyricetin protects neurons in an MPTP-induced model of Parkinson's disease by suppressing glycogen synthase kinase-3 beta activity.

Acta Pharmacol Sin. 2016-9

[10]
Protective Effects of Dihydromyricetin against •OH-Induced Mesenchymal Stem Cells Damage and Mechanistic Chemistry.

Molecules. 2016-5-9

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