泊沙康唑诱发的表观盐皮质激素过多症。 - Suppr

Posaconazole-Induced Apparent Mineralocorticoid Excess.

作者信息

Pandit Amar, Schlondorff Johannes

机构信息

Division of Nephrology, Department of Medicine, Beth Israel Deaconess Medical Center and Harvard Medical School, Boston, Massachusetts, USA.

出版信息

Kidney Int Rep. 2020 Oct 1;5(12):2379-2382. doi: 10.1016/j.ekir.2020.09.025. eCollection 2020 Dec.

DOI:10.1016/j.ekir.2020.09.025

PMID:33305136

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7710826/

Abstract

摘要

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b6b8/7710826/85d0d887d023/gr1.jpg

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Posaconazole-Induced Apparent Mineralocorticoid Excess.泊沙康唑诱发的表观盐皮质激素过多症。

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Refractory Hypokalemia from Syndrome of Apparent Mineralocorticoid Excess on Low-Dose Posaconazole.低剂量泊沙康唑治疗所致表观盐皮质激素过多综合征引起的难治性低钾血症

Antimicrob Agents Chemother. 2018 Jun 26;62(7). doi: 10.1128/AAC.02605-17. Print 2018 Jul.

Posaconazole-Induced Hypertension Due to Inhibition of 11-Hydroxylase and 11-Hydroxysteroid Dehydrogenase 2.泊沙康唑因抑制11β-羟化酶和11β-羟类固醇脱氢酶2而导致高血压。

J Endocr Soc. 2019 Jun 6;3(7):1361-1366. doi: 10.1210/js.2019-00189. eCollection 2019 Jul 1.

Molecular mechanisms of posaconazole- and itraconazole-induced pseudohyperaldosteronism and assessment of other systemically used azole antifungals.泊沙康唑和伊曲康唑诱导的假性醛固酮增多症的分子机制，以及其他系统使用的唑类抗真菌药物的评估。

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11β-Hydroxysteroid Dehydrogenase Inhibition in Posaconazole-Induced Hypertension and Hypokalemia.泊沙康唑诱导的高血压和低钾血症中11β-羟基类固醇脱氢酶的抑制作用

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Posaconazole-induced hypertension and hypokalemia due to inhibition of the 11β-hydroxylase enzyme.泊沙康唑因抑制11β-羟化酶而导致高血压和低钾血症。

Clin Kidney J. 2018 Oct;11(5):691-693. doi: 10.1093/ckj/sfx156. Epub 2018 Jan 30.

Posaconazole-Induced Pseudohyperaldosteronism.泊沙康唑致假性醛固酮增多症。

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Liquorice-induced sodium retention. Merely an acquired condition of apparent mineralocorticoid excess? A case report.

Ann Ital Med Int. 2000 Oct-Dec;15(4):296-300.

Hereditary causes of primary aldosteronism and other disorders of apparent excess mineralocorticoid activity.原发性醛固酮增多症及其他明显的盐皮质激素活性过高相关疾病的遗传病因。

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Apparent mineralocorticoid excess syndrome: report of one family with three affected children.表观盐皮质激素过多综合征：一个有三名患病儿童的家庭报告。

J Pediatr Endocrinol Metab. 2012;25(11-12):1083-8. doi: 10.1515/jpem-2012-0113.

引用本文的文献

Antifungal Therapy with Azoles Induced the Syndrome of Acquired Apparent Mineralocorticoid Excess: a Literature and Database Analysis.唑类抗真菌药物治疗诱导获得性表观盐皮质激素过多症：文献和数据库分析。

Antimicrob Agents Chemother. 2022 Jan 18;66(1):e0166821. doi: 10.1128/AAC.01668-21. Epub 2021 Oct 18.

本文引用的文献

11β-Hydroxysteroid Dehydrogenase Inhibition in Posaconazole-Induced Hypertension and Hypokalemia.泊沙康唑诱导的高血压和低钾血症中11β-羟基类固醇脱氢酶的抑制作用

Antimicrob Agents Chemother. 2017 Jul 25;61(8). doi: 10.1128/AAC.00760-17. Print 2017 Aug.

Inhibition of 11β-hydroxysteroid dehydrogenase 2 by the fungicides itraconazole and posaconazole.抗真菌药伊曲康唑和泊沙康唑对11β-羟基类固醇脱氢酶2的抑制作用。

Biochem Pharmacol. 2017 Apr 15;130:93-103. doi: 10.1016/j.bcp.2017.01.010. Epub 2017 Jan 25.

Pharmacologic and clinical evaluation of posaconazole.泊沙康唑的药理学与临床评价

Expert Rev Clin Pharmacol. 2015 May;8(3):321-34. doi: 10.1586/17512433.2015.1034689.

[Itraconazole: a new drug-related cause of hypertension].伊曲康唑：一种新的药物相关性高血压病因

Ann Cardiol Angeiol (Paris). 2014 Jun;63(3):213-5. doi: 10.1016/j.ancard.2014.05.007. Epub 2014 Jun 2.

Posaconazole exposure-response relationship: evaluating the utility of therapeutic drug monitoring.泊沙康唑暴露-反应关系：评估治疗药物监测的效用。

Antimicrob Agents Chemother. 2012 Jun;56(6):2806-13. doi: 10.1128/AAC.05900-11. Epub 2012 Mar 5.

Congenital deficiency of 11beta-hydroxysteroid dehydrogenase (apparent mineralocorticoid excess syndrome): diagnostic value of urinary free cortisol and cortisone.11β-羟类固醇脱氢酶先天性缺乏（表观盐皮质激素过多综合征）：尿游离皮质醇和可的松的诊断价值

J Endocrinol Invest. 2001 Jan;24(1):17-23. doi: 10.1007/BF03343803.

Localisation of 11 beta-hydroxysteroid dehydrogenase--tissue specific protector of the mineralocorticoid receptor.11β-羟类固醇脱氢酶的定位——盐皮质激素受体的组织特异性保护因子

Lancet. 1988 Oct 29;2(8618):986-9. doi: 10.1016/s0140-6736(88)90742-8.

Mineralocorticoid action: target tissue specificity is enzyme, not receptor, mediated.盐皮质激素作用：靶组织特异性是由酶介导而非受体介导的。

Science. 1988 Oct 28;242(4878):583-5. doi: 10.1126/science.2845584.

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Posaconazole-Induced Apparent Mineralocorticoid Excess.

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