Scott I, Parkes R, Cameron D P
Princess Alexandra Hospital, Woolloongabba, QLD.
Med J Aust. 1988 Jan 18;148(2):94-6. doi: 10.5694/j.1326-5377.1988.tb104520.x.
This case report describes a patient who presented with severe biventricular cardiac failure and shock, whose cardiac function returned to normal after the removal of a noradrenaline-secreting benign phaeochromocytoma. A catecholamine-induced cardiomyopathy with cardiogenic shock, rather than catecholamine-mediated peripheral vasoconstriction alone, is postulated as a mechanism to account for the marked hypoperfusion.
本病例报告描述了一名出现严重双心室心力衰竭和休克的患者,在切除分泌去甲肾上腺素的良性嗜铬细胞瘤后,其心功能恢复正常。推测一种儿茶酚胺诱导的心肌病伴心源性休克,而非单纯儿茶酚胺介导的外周血管收缩,是导致明显灌注不足的机制。