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整合 SNP 疾病关联、eQTL 和富集分析以鉴定慢性阻塞性肺疾病的风险 SNP 和易感基因。

Integration of SNP Disease Association, eQTL, and Enrichment Analyses to Identify Risk SNPs and Susceptibility Genes in Chronic Obstructive Pulmonary Disease.

机构信息

College of Bioinformatics Science and Technology, Harbin Medical University, Harbin, China.

Department of Respiratory, The Second Affiliated Hospital, Harbin Medical University, Harbin, China.

出版信息

Biomed Res Int. 2020 Dec 29;2020:3854196. doi: 10.1155/2020/3854196. eCollection 2020.

DOI:10.1155/2020/3854196
PMID:33457407
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC7785362/
Abstract

Chronic obstructive pulmonary disease (COPD) is a complex disease caused by the disturbance of genetic and environmental factors. Single-nucleotide polymorphisms (SNPs) play a vital role in the genetic dissection of complex diseases. In-depth analysis of SNP-related information could recognize disease-associated biomarkers and further uncover the genetic mechanism of complex diseases. Risk-related variants might act on the disease by affecting gene expression and gene function. Through integrating SNP disease association study and expression quantitative trait loci (eQTL) analysis, as well as functional enrichment of containing known causal genes, four risk SNPs and four corresponding susceptibility genes were identified utilizing next-generation sequencing (NGS) data of COPD. Of the four risk SNPs, one could be found in the SNPedia database that stored disease-related SNPs and has been linked to a disease in the literature. Four genes showed significant differences from the perspective of normal/disease or variant/nonvariant samples, as well as the high performance of sample classification. It is speculated that the four susceptibility genes could be used as biomarkers of COPD. Furthermore, three of our susceptibility genes have been confirmed in the literature to be associated with COPD. Among them, two genes had an impact on the significance of expression correlation of known causal genes they interact with, respectively. Overall, this research may present novel insights into the diagnosis and pathogenesis of COPD and susceptibility gene identification of other complex diseases.

摘要

慢性阻塞性肺疾病(COPD)是一种由遗传和环境因素紊乱引起的复杂疾病。单核苷酸多态性(SNPs)在复杂疾病的遗传剖析中起着至关重要的作用。深入分析 SNP 相关信息可以识别疾病相关的生物标志物,并进一步揭示复杂疾病的遗传机制。风险相关变体可能通过影响基因表达和基因功能来作用于疾病。通过整合 SNP 疾病关联研究和表达数量性状基因座(eQTL)分析,以及包含已知因果基因的功能富集,利用 COPD 的下一代测序(NGS)数据,确定了四个风险 SNP 和四个相应的易感基因。在四个风险 SNP 中,有一个可以在 SNPedia 数据库中找到,该数据库存储了与疾病相关的 SNP,并在文献中与疾病相关联。从正常/疾病或变体/非变体样本的角度来看,四个基因表现出显著差异,以及样本分类的高性能。据推测,这四个易感基因可以用作 COPD 的生物标志物。此外,我们的四个易感基因中有三个已在文献中证实与 COPD 相关。其中,两个基因分别对其相互作用的已知因果基因表达相关性的显著性产生了影响。总的来说,这项研究可能为 COPD 的诊断和发病机制以及其他复杂疾病的易感基因识别提供新的见解。

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