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T 细胞来源的细胞因子包含外泌体诱导口腔扁平苔藓角质形成细胞凋亡。

T cell-derived exosomes containing cytokines induced keratinocytes apoptosis in oral lichen planus.

机构信息

The State Key Laboratory Breeding Base of Basic Science of Stomatology (Hubei-MOST) and Key Laboratory of Oral Biomedicine Ministry of Education, School and Hospital of Stomatology, Wuhan University, Wuhan, China.

Department of Oral Medicine, School and Hospital of Stomatology, Wuhan University, Wuhan, China.

出版信息

Oral Dis. 2022 Apr;28(3):682-690. doi: 10.1111/odi.13795. Epub 2021 Feb 16.

Abstract

OBJECTIVE

Oral lichen planus (OLP) is a T cell-mediated inflammatory disease with uncertain etiology. Exosomes are cell-derived vesicles containing biological cargo, being associated with the development of multiple inflammatory diseases. The present study aims to investigate the role of T cell-derived exosomes in the pathogenesis of OLP.

METHODS

Exosomal marker CD63 was detected in OLP lesions by immunohistochemistry. Twenty-three cytokines in T cell-derived exosomes were assessed using luminex xMAP-based assay. After co-incubating with exosomes, the apoptosis of keratinocytes and the proliferation of Jurkat cells were assessed via flow cytometry and cell counting kit-8 assay, respectively.

RESULTS

CD63 was highly expressed in the lymphocyte infiltrated areas of OLP lesions. OLP T cell-derived exosomes contained upregulated interleukin-7, -10, -12, -17 and downregulated interleukin-1β, -5, and interferon-γ. Both exosomes from OLP patients and controls induced the apoptosis of keratinocytes and altered their morphology. Moreover, healthy control-derived exosomes markedly inhibited the proliferation of Jurkat cells, whereas OLP-derived exosomes exhibited no inhibitory effect.

CONCLUSIONS

OLP T cell-derived exosomes have an aberrant cytokine profile and could trigger the apoptosis of keratinocytes in vitro, which may be involved in the pathogenesis of OLP.

摘要

目的

口腔扁平苔藓(OLP)是一种以 T 细胞介导的炎症性疾病,病因不明。外泌体是含有生物货物的细胞衍生小泡,与多种炎症性疾病的发展有关。本研究旨在探讨 T 细胞来源的外泌体在 OLP 发病机制中的作用。

方法

通过免疫组织化学检测 OLP 病变中外泌体标志物 CD63。采用基于 Luminex xMAP 的检测方法评估 T 细胞来源的外泌体中的 23 种细胞因子。通过共孵育外泌体,通过流式细胞术和细胞计数试剂盒-8 检测分别评估角质形成细胞的凋亡和 Jurkat 细胞的增殖。

结果

CD63 在 OLP 病变的淋巴细胞浸润区高表达。OLP T 细胞来源的外泌体中白细胞介素-7、-10、-12、-17 上调,白细胞介素-1β、-5 和干扰素-γ下调。来自 OLP 患者和对照组的外泌体均可诱导角质形成细胞凋亡并改变其形态。此外,健康对照组来源的外泌体显著抑制 Jurkat 细胞的增殖,而 OLP 来源的外泌体则无抑制作用。

结论

OLP T 细胞来源的外泌体具有异常的细胞因子谱,并可在体外触发角质形成细胞凋亡,这可能与 OLP 的发病机制有关。

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