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核心技术专利：CN118964589B侵权必究

核心技术专利：CN118964589B侵权必究

Department of Biochemistry C, Panum Institute, University of Copenhagen, Denmark.

Int J Biochem. 1988;20(3):309-15. doi: 10.1016/0020-711x(88)90356-4.

Negatively charged phospholipids promote initiation of the contact activation system in the blood coagulation. 2. Neutral phospholipids were unable to activate this system. 3. The activation is inhibited by beta 2-glycoprotein I at physiological concentrations. 4. The results raise the question whether people with low concentration of beta 2-glycoprotein I are more easily exposed to blood coagulation defects, such as disseminated intravascular coagulation, than those with normal concentration of beta 2-glycoprotein I.

带负电荷的磷脂促进血液凝固中接触激活系统的启动。2. 中性磷脂无法激活该系统。3. 在生理浓度下，β2-糖蛋白I可抑制这种激活。4. 这些结果提出了一个问题，即与β2-糖蛋白I浓度正常的人相比，β2-糖蛋白I浓度低的人是否更容易出现凝血缺陷，如弥散性血管内凝血。

Department of Biochemistry C, Panum Institute, University of Copenhagen, Denmark.

Int J Biochem. 1988;20(3):309-15. doi: 10.1016/0020-711x(88)90356-4.

Negatively charged phospholipids promote initiation of the contact activation system in the blood coagulation. 2. Neutral phospholipids were unable to activate this system. 3. The activation is inhibited by beta 2-glycoprotein I at physiological concentrations. 4. The results raise the question whether people with low concentration of beta 2-glycoprotein I are more easily exposed to blood coagulation defects, such as disseminated intravascular coagulation, than those with normal concentration of beta 2-glycoprotein I.

带负电荷的磷脂促进血液凝固中接触激活系统的启动。2. 中性磷脂无法激活该系统。3. 在生理浓度下，β2-糖蛋白I可抑制这种激活。4. 这些结果提出了一个问题，即与β2-糖蛋白I浓度正常的人相比，β2-糖蛋白I浓度低的人是否更容易出现凝血缺陷，如弥散性血管内凝血。