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Soluble receptor for advanced glycation end products (sRAGE) and asthma: Mendelian randomisation study.

作者信息

Raita Yoshihiko, Zhu Zhaozhong, Freishtat Robert J, Fujiogi Michimasa, Liang Liming, Patregnani Jason T, Camargo Carlos A, Hasegawa Kohei

机构信息

Department of Emergency Medicine, Massachusetts General Hospital, Harvard Medical School, Boston, MA, USA.

Division of Emergency Medicine, Children's National Hospital, Washington, DC, USA.

出版信息

Pediatr Allergy Immunol. 2021 Jul;32(5):1100-1103. doi: 10.1111/pai.13478. Epub 2021 Mar 5.

DOI:10.1111/pai.13478
PMID:33599351
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8249337/
Abstract
摘要

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A genomic approach identifies sRAGE as a putatively causal protein for asthma.一种基因组学方法将可溶性晚期糖基化终末产物受体鉴定为哮喘的一种可能的因果蛋白。
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Am J Physiol Lung Cell Mol Physiol. 2019 Jun 1;316(6):L1061-L1069. doi: 10.1152/ajplung.00359.2018. Epub 2019 Mar 6.
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Genes (Basel). 2024 Dec 17;15(12):1608. doi: 10.3390/genes15121608.
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sRAGE levels are decreased in plasma and sputum of COPD secondary to biomass-burning smoke and tobacco smoking: Differences according to the rs3134940 AGER variant.在因生物质燃烧烟雾和吸烟导致的慢性阻塞性肺疾病（COPD）患者的血浆和痰液中，可溶性晚期糖基化终末产物受体（sRAGE）水平降低：根据AGER基因rs3134940变体存在差异。
Heliyon. 2024 Mar 22;10(7):e28675. doi: 10.1016/j.heliyon.2024.e28675. eCollection 2024 Apr 15.
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本文引用的文献

1
Serum Soluble Receptor for Advanced Glycation End Products in Infants With Bronchiolitis: Associations With Acute Severity and Recurrent Wheeze.毛细支气管炎婴儿血清晚期糖基化终产物可溶性受体：与急性严重程度和反复喘息的相关性。
Clin Infect Dis. 2021 Nov 2;73(9):e2665-e2672. doi: 10.1093/cid/ciaa1700.
2
Genomic and drug target evaluation of 90 cardiovascular proteins in 30,931 individuals.对 30931 个人的 90 种心血管蛋白进行基因组和药物靶点评估。
Nat Metab. 2020 Oct;2(10):1135-1148. doi: 10.1038/s42255-020-00287-2. Epub 2020 Oct 16.
3
The axis of the receptor for advanced glycation endproducts in asthma and allergic airway disease.
Serum sRAGE levels in children with atopic dermatitis: a prospective study.
特应性皮炎患儿血清可溶性晚期糖基化终末产物受体水平：一项前瞻性研究。
Postepy Dermatol Alergol. 2023 Dec;40(6):766-771. doi: 10.5114/ada.2023.133585. Epub 2024 Jan 8.
4
Do advanced glycation end products contribute to food allergy?晚期糖基化终产物是否会导致食物过敏？
Front Allergy. 2023 Apr 4;4:1148181. doi: 10.3389/falgy.2023.1148181. eCollection 2023.
5
RAGE contributes to allergen driven severe neutrophilic airway inflammation NLRP3 inflammasome activation in mice.RAGE 导致变应原驱动的严重中性粒细胞性气道炎症和小鼠 NLRP3 炎性体激活。
Front Immunol. 2023 Jan 26;14:1039997. doi: 10.3389/fimmu.2023.1039997. eCollection 2023.
6
The causal relationship between allergic diseases and heart failure: Evidence from Mendelian randomization study.过敏疾病与心力衰竭之间的因果关系：来自孟德尔随机化研究的证据。
PLoS One. 2022 Jul 29;17(7):e0271985. doi: 10.1371/journal.pone.0271985. eCollection 2022.
7
A genomic approach identifies sRAGE as a putatively causal protein for asthma.一种基因组学方法将可溶性晚期糖基化终末产物受体鉴定为哮喘的一种可能的因果蛋白。
J Allergy Clin Immunol. 2022 Jun;149(6):1992-1997.e12. doi: 10.1016/j.jaci.2021.11.027. Epub 2021 Dec 30.
8
Nasopharyngeal metatranscriptome profiles of infants with bronchiolitis and risk of childhood asthma: a multicentre prospective study.婴儿毛细支气管炎与儿童哮喘风险的鼻咽转录组特征：一项多中心前瞻性研究。
Eur Respir J. 2022 Jul 13;60(1). doi: 10.1183/13993003.02293-2021. Print 2022 Jul.
哮喘和过敏性气道疾病中晚期糖基化终产物受体的轴。
Allergy. 2021 May;76(5):1350-1366. doi: 10.1111/all.14600. Epub 2020 Oct 9.
4
Shared genetic and experimental links between obesity-related traits and asthma subtypes in UK Biobank.英国生物库中肥胖相关特征与哮喘亚型之间的共享遗传和实验关联。
J Allergy Clin Immunol. 2020 Feb;145(2):537-549. doi: 10.1016/j.jaci.2019.09.035. Epub 2019 Oct 24.
5
Shared genetics of asthma and mental health disorders: a large-scale genome-wide cross-trait analysis.哮喘和精神健康障碍的共享遗传学：大规模全基因组跨表型分析。
Eur Respir J. 2019 Dec 19;54(6). doi: 10.1183/13993003.01507-2019. Print 2019 Dec.
6
Reading Mendelian randomisation studies: a guide, glossary, and checklist for clinicians.阅读孟德尔随机化研究：临床医生指南、词汇表和清单。
BMJ. 2018 Jul 12;362:k601. doi: 10.1136/bmj.k601.
7
A genome-wide cross-trait analysis from UK Biobank highlights the shared genetic architecture of asthma and allergic diseases.英国生物库的全基因组跨性状分析强调了哮喘和过敏性疾病的共享遗传结构。
Nat Genet. 2018 Jun;50(6):857-864. doi: 10.1038/s41588-018-0121-0. Epub 2018 May 21.
8
Decreased soluble RAGE in neutrophilic asthma is correlated with disease severity and RAGE G82S variants.中性粒细胞性哮喘患者可溶性 RAGE 减少与疾病严重程度和 RAGE G82S 变异体相关。
Mol Med Rep. 2018 Mar;17(3):4131-4137. doi: 10.3892/mmr.2017.8302. Epub 2017 Dec 18.
9
sRAGE alleviates neutrophilic asthma by blocking HMGB1/RAGE signalling in airway dendritic cells.sRAGE 通过阻断气道树突状细胞中的 HMGB1/RAGE 信号通路缓解中性粒细胞性哮喘。
Sci Rep. 2017 Oct 27;7(1):14268. doi: 10.1038/s41598-017-14667-4.
10
Pulmonary receptor for advanced glycation end-products promotes asthma pathogenesis through IL-33 and accumulation of group 2 innate lymphoid cells.晚期糖基化终产物的肺受体通过白细胞介素-33和2型固有淋巴细胞的积累促进哮喘发病机制。
J Allergy Clin Immunol. 2015 Sep;136(3):747-756.e4. doi: 10.1016/j.jaci.2015.03.011. Epub 2015 Apr 28.