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一名接受减压和经皮椎弓根螺钉固定术的患者,其胸椎脊髓型颈椎病可能由弥漫性特发性骨肥厚引起。

Thoracic spondylotic myelopathy presumably caused by diffuse idiopathic skeletal hyperostosis in a patient who underwent decompression and percutaneous pedicle screw fixation.

作者信息

Miyoshi Shota, Morino Tadao, Takeda Haruhiko, Nakata Hiroshi, Hino Masayuki, Misaki Hiroshi, Murakami Yusuke, Imai Hiroshi, Miura Hiromasa

机构信息

Department of Orthopedic Surgery, Saijo Central Hospital, Saijo, Japan.

Department of Orthopedic Surgery, Ehime University School of Medicine, Tohon, Japan.

出版信息

SAGE Open Med Case Rep. 2021 Jan 21;9:2050313X20987796. doi: 10.1177/2050313X20987796. eCollection 2021.

Abstract

A 74-year-old man developed bilateral lower limb spastic paresis. He was diagnosed with thoracic spondylotic myelopathy presumably caused by mechanical stress that was generated in the intervertebral space (T1-T2) between a vertebral bone bridge (C5-T1) due to diffuse idiopathic skeletal hyperostosis after anterior fixation of the lower cervical spine and a vertebral bone bridge (T2-T7) due to diffuse idiopathic skeletal hyperostosis in the upper thoracic spine. Treatment included posterior decompression (T1-T2 laminectomy) and percutaneous pedicle screw fixation at the C7-T4 level. Six months after surgery, the patient could walk with a cane, and the vertebral bodies T1-T2 were bridged without bone grafting. For thoracic spondylotic myelopathy associated with diffuse idiopathic skeletal hyperostosis, decompression and percutaneous pedicle screw fixation are effective therapies.

摘要

一名74岁男性出现双侧下肢痉挛性轻瘫。他被诊断为胸椎脊髓型颈椎病,推测是由于下颈椎前路固定后因弥漫性特发性骨肥厚在椎体骨桥(C5 - T1)与上胸椎椎体骨桥(T2 - T7)之间的椎间隙(T1 - T2)产生的机械应力所致。治疗包括后路减压(T1 - T2椎板切除术)以及C7 - T4节段经皮椎弓根螺钉固定。术后6个月,患者可借助拐杖行走,T1 - T2椎体无需植骨已形成骨桥。对于与弥漫性特发性骨肥厚相关的胸椎脊髓型颈椎病,减压和经皮椎弓根螺钉固定是有效的治疗方法。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/d5a1/7829458/94cc42df99f4/10.1177_2050313X20987796-fig1.jpg

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