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棘轮机制选择性导致主动固定冠状静脉窦导线特发性大移位:一例报告

Ratchet mechanism selectively causing idiopathic macrodislodgement of an active-fixation coronary sinus lead: a case report.

作者信息

Golzio Pier Giorgio, Bissolino Arianna, Ceci Raffaele, Frea Simone

机构信息

Division of Cardiology, Department of Internal Medicine, Azienda Ospedaliero-Universitaria Città della Salute e della Scienza di Torino and University of Turin, corso Bramante 88-10126 Turin, Italy.

出版信息

Eur Heart J Case Rep. 2020 Dec 28;4(6):1-6. doi: 10.1093/ehjcr/ytaa466. eCollection 2020 Dec.

Abstract

BACKGROUND

'Idiopathic' lead macrodislodgement may be due to Twiddler's syndrome depending on active twisting of pulse generator within subcutaneous pocket. All leads are involved, at any time from implantation, and frequently damaged. In the past few years, a reel syndrome was also observed: retraction of pacemaker leads into pocket without patient manipulation, owing to lead circling the generator. In other cases, a 'ratchet' mechanism has been postulated. Reel and ratchet mechanisms require loose anchoring, occur generally briefly after implantation, with non-damaged leads. We report the first case of an active-fixation coronary sinus lead selective macrodislodgement involving such ratchet mechanism.

CASE SUMMARY

A 65-year-old man underwent biventricular defibrillator device implantation, with active-fixation coronary sinus lead. Eight months later, he complained of muscle contractions over device pocket. At fluoroscopy, coronary sinus lead was found near to pocket, outside of thoracic inlet. Atrial and ventricular leads were in normal position. After opening pocket, a short tract of coronary sinus lead appeared anteriorly dislocated to generator, while greater length of lead body twisted a reel behind. The distal part of lead was found outside venous entry at careful dissection. Atrial and ventricular leads were firmly anchored.

DISCUSSION

Our case is a selective 'Idiopathic' lead macrodislodgement, possibly due to a ratchet mechanism between the lead and the suture sleeve, induced by normal arm motion; such mechanism incredibly, and for first time in literature involves a coronary sinus active-fixation lead.

CONCLUSION

Careful attention should always be paid to secure anchoring even of active-fixation coronary sinus leads.

摘要

背景

“特发性”导线大移位可能是由于旋弄综合征,这取决于皮下囊袋内脉冲发生器的主动扭转。所有导线在植入后的任何时候都可能受累,并且经常受损。在过去几年中,还观察到一种卷轴综合征:起搏器导线在没有患者操作的情况下回缩到囊袋中,这是由于导线环绕发生器所致。在其他情况下,有人提出了“棘轮”机制。卷轴和棘轮机制需要固定不牢,通常在植入后不久发生,导线未受损。我们报告了首例涉及这种棘轮机制的主动固定冠状静脉窦导线选择性大移位病例。

病例摘要

一名65岁男性接受了双心室除颤器植入术,采用主动固定冠状静脉窦导线。八个月后,他抱怨装置囊袋上方的肌肉收缩。在荧光透视检查中,发现冠状静脉窦导线位于囊袋附近,胸廓入口之外。心房和心室导线位置正常。打开囊袋后,发现冠状静脉窦导线的一小段向前移位至发生器,而导线体的更长部分在后面扭成了一个卷轴。仔细解剖后发现导线远端在静脉入口之外。心房和心室导线固定牢固。

讨论

我们的病例是一种选择性“特发性”导线大移位,可能是由于正常手臂运动导致导线与缝合套之间的棘轮机制引起的;令人难以置信的是,这种机制在文献中首次涉及冠状静脉窦主动固定导线。

结论

即使对于冠状静脉窦主动固定导线,也应始终注意确保牢固固定。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7fd5/7891260/6c5d49838fad/ytaa466f1.jpg

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