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Acyclovir as a Novel Treatment for Severe Chronic Active Epstein-Barr Virus.阿昔洛韦作为严重慢性活动性爱泼斯坦-巴尔病毒的新型治疗方法。
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Epstein-Barr Virus Hepatitis Masquerading as Painless Jaundice.伪装成无痛性黄疸的爱泼斯坦-巴尔病毒肝炎
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Adenosine deaminase as a marker for the severity of infectious mononucleosis secondary to EBV in children.腺苷脱氨酶作为儿童 EB 病毒感染性单核细胞增多症严重程度的标志物。
BMC Infect Dis. 2022 Feb 21;22(1):164. doi: 10.1186/s12879-022-07150-7.
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Epstein-Barr Virus-induced Jaundice.爱泼斯坦-巴尔病毒引起的黄疸
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Acute cholestatic hepatitis induced by Epstein-Barr virus infection in an adult: a case report.成人感染爱泼斯坦-巴尔病毒所致急性胆汁淤积性肝炎：一例报告
J Med Case Rep. 2016 Mar 27;10:75. doi: 10.1186/s13256-016-0859-x.
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Clinical characteristics of primary Epstein Barr virus hepatitis with elevation of alkaline phosphatase and γ-glutamyltransferase in children.儿童原发性 EBV 肝炎伴碱性磷酸酶和 γ-谷氨酰转移酶升高的临床特征。
Yonsei Med J. 2014 Jan;55(1):107-12. doi: 10.3349/ymj.2014.55.1.107.

本文引用的文献

1
Complement deficiency states and infection: epidemiology, pathogenesis and consequences of neisserial and other infections in an immune deficiency.补体缺陷状态与感染：免疫缺陷状态下奈瑟菌及其他感染的流行病学、发病机制及后果
Medicine (Baltimore). 1984 Sep;63(5):243-73.
2
Infectious mononucleosis hepatitis.传染性单核细胞增多症肝炎
Semin Liver Dis. 1984 Nov;4(4):301-6. doi: 10.1055/s-2008-1040659.
3
Infectious mononucleosis complicated by hepatic coma.传染性单核细胞增多症并发肝昏迷。
Am J Gastroenterol. 1969 Mar;51(3):200-7.
4
Immune response of a patient with deficiency of the fourth component of complement and systemic lupus erythematosus.一名补体第四成分缺乏症患者与系统性红斑狼疮的免疫反应。
N Engl J Med. 1979 May 17;300(20):1124-9. doi: 10.1056/NEJM197905173002002.
5
A new manifestation of infection with Epstein-Barr virus.爱泼斯坦-巴尔病毒感染的一种新表现形式。
Br Med J. 1978 Jul 22;2(6132):248. doi: 10.1136/bmj.2.6132.248.

肝功能衰竭与爱泼斯坦-巴尔病毒感染

Liver failure and Epstein-Barr virus infection.

作者信息

Shaw N J, Evans J H

机构信息

Department of Paediatrics and Child Health, St James's University Hospital, Leeds.

出版信息

Arch Dis Child. 1988 Apr;63(4):432-3. doi: 10.1136/adc.63.4.432.

DOI:10.1136/adc.63.4.432
PMID:3365013
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1778815/
Abstract

A 5 year old boy developed liver failure secondary to infection with Epstein-Barr virus. He was subsequently shown to have a partial C4 complement deficiency. The importance of considering Epstein-Barr virus as a cause of fulminant hepatic failure and the need to assess immune state in such an event is emphasised.

摘要

一名5岁男孩因感染爱泼斯坦-巴尔病毒继发肝衰竭。随后发现他存在部分C4补体缺乏。强调了将爱泼斯坦-巴尔病毒视为暴发性肝衰竭病因的重要性以及在此类情况下评估免疫状态的必要性。