自身免疫性慢性活动性肝炎伴严重高球蛋白血症中的非精神性原发性烦渴症
Non-psychogenic primary polydipsia in autoimmune chronic active hepatitis with severe hyperglobulinaemia.
作者信息
Tobin M V, Morris A I
机构信息
Gastroenterology Unit, Royal Liverpool Hospital.
出版信息
Gut. 1988 Apr;29(4):548-9. doi: 10.1136/gut.29.4.548.
Abstract
The association of hyperglobulinaemia with renal tubular acidosis and nephrogenic diabetes insipidus is well established. A patient with marked hyperglobulinaemia due to autoimmune chronic active hepatitis is described who presented with severe polydipsia and polyuria but had entirely normal renal tubular function indicating a primary thirst disorder.
摘要
高球蛋白血症与肾小管酸中毒及肾性尿崩症之间的关联已得到充分证实。本文描述了一名因自身免疫性慢性活动性肝炎导致明显高球蛋白血症的患者，该患者表现出严重的烦渴和多尿，但肾小管功能完全正常，提示存在原发性口渴障碍。
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引用本文的文献
1
Primary polydipsia and autoimmune chronic active hepatitis.原发性烦渴与自身免疫性慢性活动性肝炎。
Gut. 1988 Dec;29(12):1755. doi: 10.1136/gut.29.12.1755.
本文引用的文献
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Hyperglobulinaemic renal tubular acidosis: a report of nine cases.高球蛋白血症性肾小管酸中毒：9例报告
Br Med J. 1970 Jul 18;3(5715):143-6. doi: 10.1136/bmj.3.5715.143.
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