CAPHRI School for Public Health and Primary Care, Faculty of Health, Medicine, and Life Sciences, Maastricht University, 6211 LM Maastricht, The Netherlands.
Private Practice, Pleasanton, CA 94565, USA.
Int J Environ Res Public Health. 2021 Mar 12;18(6):2901. doi: 10.3390/ijerph18062901.
Injury claims associated with minimal damage rear impact traffic crashes are often defended using a "biomechanical approach," in which the occupant forces of the crash are compared to the forces of activities of daily living (ADLs), resulting in the conclusion that the risk of injury from the crash is the same as for ADLs. The purpose of the present investigation is to evaluate the scientific validity of the central operating premise of the biomechanical approach to injury causation; that occupant acceleration is a scientifically valid proxy for injury risk. Data were abstracted, pooled, and compared from three categories of published literature: (1) volunteer rear impact crash testing studies, (2) ADL studies, and (3) observational studies of real-world rear impacts. We compared the occupant accelerations of minimal or no damage (i.e., 3 to 11 kph speed change or "delta V") rear impact crash tests to the accelerations described in 6 of the most commonly reported ADLs in the reviewed studies. As a final step, the injury risk observed in real world crashes was compared to the results of the pooled crash test and ADL analyses, controlling for delta V. The results of the analyses indicated that average peak linear and angular acceleration forces observed at the head during rear impact crash tests were typically at least several times greater than average forces observed during ADLs. In contrast, the injury risk of real-world minimal damage rear impact crashes was estimated to be at least 2000 times greater than for any ADL. The results of our analysis indicate that the principle underlying the biomechanical injury causation approach, that occupant acceleration is a proxy for injury risk, is scientifically invalid. The biomechanical approach to injury causation in minimal damage crashes invariably results in the vast underestimation of the actual risk of such crashes, and should be discontinued as it is a scientifically invalid practice.
与轻微后部碰撞事故相关的伤害索赔通常采用“生物力学方法”进行辩护,该方法将碰撞中的乘员力与日常生活活动(ADL)的力进行比较,得出的结论是,碰撞造成伤害的风险与 ADL 相同。本研究的目的是评估生物力学伤害因果关系方法的核心操作前提的科学有效性;即乘员加速度是伤害风险的科学有效替代物。从已发表文献的三个类别中提取、汇总和比较数据:(1)志愿者后部碰撞碰撞测试研究;(2)ADL 研究;(3)现实世界后部碰撞的观察性研究。我们将最小或无损伤(即 3 至 11 公里/小时的速度变化或“Delta V”)的后部碰撞测试中的乘员加速度与 6 项研究中最常报告的 ADL 中描述的加速度进行了比较。作为最后一步,控制 Delta V 后,将现实世界碰撞中的伤害风险与汇总的碰撞测试和 ADL 分析结果进行了比较。分析结果表明,在后部碰撞测试中头部观察到的平均峰值线性和角加速度力通常至少是 ADL 中观察到的力的几倍。相比之下,现实世界中轻微损坏的后部碰撞事故的伤害风险估计至少比任何 ADL 高出 2000 倍。我们的分析结果表明,生物力学伤害因果关系方法的基本原则,即乘员加速度是伤害风险的替代物,在科学上是无效的。在轻微损坏的碰撞中,生物力学伤害因果关系方法必然会导致对这种碰撞实际风险的极大低估,并且应该停止使用,因为它是一种在科学上无效的做法。