Department of Stroke Neurology, Kohnan Hospital, Japan.
Intern Med. 2021 Oct 1;60(19):3161-3166. doi: 10.2169/internalmedicine.6994-21. Epub 2021 Apr 19.
A 68-year-old man was admitted to our department because of left incomplete homonymous hemianopia accompanied by hyperglycemia. Both T2-weighted and diffusion-weighted imaging revealed a low signal intensity along the subcortex and high signal intensity along the cortex on the right parietal and occipital lobes. Furthermore, arterial spin labeling and single-photon emission computed tomography showed hyperperfusion at the right parieto-occipital lobe. However, the electroencephalography result was normal. Hyperperfusion improved after controlling the blood glucose levels; nevertheless, homonymous hemianopia remained. We suspect that the irreversible brain damage was attributable to hyperperfusion associated with long-term hyperglycemia.
一位 68 岁男性因左不完全面盲伴高血糖症被收入我科。T2 加权像和弥散加权成像显示右侧顶叶和枕叶皮质及皮质下均呈低信号,而呈高信号。此外,动脉自旋标记和单光子发射计算机断层扫描显示右侧顶枕叶高灌注。然而,脑电图结果正常。血糖控制后高灌注改善,但仍存在同向偏盲。我们怀疑与长期高血糖相关的高灌注导致了不可逆转的脑损伤。