Terui Toshihiro, Iwai-Takano Masumi, Watanabe Tomoyuki
Division of Internal Medicine, Health Co-op, Watari Hospital, Fukushima, Japan.
International Community Health, Graduate School of Medicine, Fukushima Medical University, Fukushima, Japan.
Case Rep Cardiol. 2021 Apr 2;2021:6637720. doi: 10.1155/2021/6637720. eCollection 2021.
This case report presents a patient with Takotsubo cardiomyopathy (TCM) and complete atrioventricular (AV) block who was treated with permanent pacemaker implantation. A 78-year-old woman with a history of hypertension presented with a 6-month history of palpitations. On initial evaluation, her heart rate was 40 beats/minute. Electrocardiography revealed a complete AV block and T-wave inversion in these leads: I, II, aVL, aVF, and V3-6. Echocardiography showed akinesis from the midventricle to the apex and hyperkinesis on the basal segments. The patient was diagnosed with TCM and complete AV block. Because improvement of TCM may subsequently improve the AV node dysfunction associated with TCM, the patient was admitted for treatment of heart failure without pacemaker implantation. The left ventricular (LV) abnormal wall motion improved gradually; however, the AV block persisted intermittently. On hospital day 14, a pause of 5-6 seconds without LV contraction was observed, and permanent pacemaker implantation was performed. On day 92, echocardiography revealed normal LV wall motion. However, electrocardiography revealed that the pacemaker rhythm with atrial sensing and ventricular pacing remained. Although specific degree of damage that may result from AV block associated with TCM is unknown, some of these patients require pacemaker implantation, despite improvement of abnormality in LV wall motion.
本病例报告介绍了一名患有Takotsubo心肌病(TCM)并伴有完全性房室传导阻滞的患者,该患者接受了永久性起搏器植入治疗。一名有高血压病史的78岁女性,出现心悸6个月。初始评估时,其心率为40次/分钟。心电图显示在I、II、aVL、aVF及V3 - 6导联存在完全性房室传导阻滞及T波倒置。超声心动图显示从心室中部至心尖运动减弱,基底部节段运动增强。该患者被诊断为TCM合并完全性房室传导阻滞。由于TCM的改善可能随后改善与TCM相关的房室结功能障碍,患者被收治以治疗心力衰竭,未植入起搏器。左心室(LV)异常壁运动逐渐改善;然而,房室传导阻滞仍间歇性存在。在住院第14天,观察到一次长达5 - 6秒的停搏且无LV收缩,遂进行了永久性起搏器植入。在第92天,超声心动图显示LV壁运动正常。然而,心电图显示仍为心房感知和心室起搏的起搏器节律。尽管与TCM相关的房室传导阻滞可能导致的具体损伤程度尚不清楚,但这些患者中的一些人尽管LV壁运动异常有所改善,仍需要植入起搏器。