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通过转录组改变和心脏重构监测来描述一个长期慢性心力衰竭模型。

Characterizing a long-term chronic heart failure model by transcriptomic alterations and monitoring of cardiac remodeling.

机构信息

Department of Cardiology, State Key Laboratory of Organ Failure Research, Guangdong Provincial Key Lab of Shock and Microcirculation, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.

Department of Oncology, Nanfang Hospital, Southern Medical University, Guangzhou 510515, China.

出版信息

Aging (Albany NY). 2021 Apr 23;13(10):13585-13614. doi: 10.18632/aging.202879.

DOI:10.18632/aging.202879
PMID:33891565
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8202904/
Abstract

The long-term characteristics of transcriptomic alterations and cardiac remodeling in chronic heart failure (CHF) induced by myocardial infarction (MI) in mice are not well elucidated. This study aimed to reveal the dynamic changes in the transcriptome and cardiac remodeling in post-MI mice over a long time period. Monitoring C57BL/6 mice with MI for 8 months showed that approximately 44% of mice died of cardiac rupture in the first 2 weeks and others survived to 8 months with left ventricular (LV) aneurysm. The transcriptomic profiling analysis of cardiac tissues showed that the Integrin and WNT pathways were activated at 8 months after MI while the metabolism-related pathways were inversely inhibited. Subsequent differential analysis at 1 and 8 months post-MI revealed significant enrichments in biological processes, including consistent regulation of metabolism-related pathways. Moreover, echocardiographic monitoring showed a progressive increase in LV dimensions and a decrease in the LV fractional shortening during the first 4 weeks, and these parameters progressed at a lower rate till 8 months. A similar trend was found in the invasive LV hemodynamics, cardiac morphological and histological analyses. These results suggested that mouse MI model is ideal for long-term studies, and transcriptomic findings may provide new CHF therapeutic targets.

摘要

心肌梗死后慢性心力衰竭(CHF)小鼠中长期转录组改变和心脏重构的特征尚不清楚。本研究旨在揭示 MI 后小鼠在长时间内转录组和心脏重构的动态变化。监测 MI 后 C57BL/6 小鼠 8 个月,结果显示约 44%的小鼠在第 2 周内因心脏破裂而死亡,其余存活至 8 个月,出现左心室(LV)瘤。心肌组织的转录组谱分析显示,MI 后 8 个月整合素和 WNT 途径被激活,而代谢相关途径则被反向抑制。随后在 MI 后 1 个月和 8 个月进行的差异分析显示,生物学过程显著富集,包括代谢相关途径的一致调节。此外,超声心动图监测显示,在最初的 4 周内,LV 尺寸逐渐增大,LV 缩短分数降低,这些参数在 8 个月内的进展速度较慢。LV 血流动力学、心脏形态学和组织学分析也发现了类似的趋势。这些结果表明,小鼠 MI 模型非常适合长期研究,转录组学发现可能为 CHF 的治疗提供新的靶点。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8d77/8202904/8f0a985b07ab/aging-13-202879-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8d77/8202904/0d1fc838b403/aging-13-202879-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8d77/8202904/839066a20f0c/aging-13-202879-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8d77/8202904/0a629d7b0cdd/aging-13-202879-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8d77/8202904/dd3365a2f0cd/aging-13-202879-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8d77/8202904/cbc8bb58d3d9/aging-13-202879-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8d77/8202904/8f0a985b07ab/aging-13-202879-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8d77/8202904/0d1fc838b403/aging-13-202879-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8d77/8202904/839066a20f0c/aging-13-202879-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8d77/8202904/0a629d7b0cdd/aging-13-202879-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8d77/8202904/dd3365a2f0cd/aging-13-202879-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8d77/8202904/cbc8bb58d3d9/aging-13-202879-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/8d77/8202904/8f0a985b07ab/aging-13-202879-g006.jpg

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