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Prdm16 通过维持内皮功能支持动脉血流恢复。

Prdm16 Supports Arterial Flow Recovery by Maintaining Endothelial Function.

机构信息

Department of Cardiovascular Sciences, Center for Molecular and Vascular Biology (S.C., J.V.W., W.D., N.C., E.A.V.J., A.Z., M.B., A.L.), KU Leuven, Leuven, Belgium.

Pharmaceutical Sciences, Physiopharmacology, University of Antwerp, Antwerp, Belgium (S.D.M., D.D.M., A.J.A.L., P.F.).

出版信息

Circ Res. 2021 Jun 25;129(1):63-77. doi: 10.1161/CIRCRESAHA.120.318501. Epub 2021 Apr 27.

DOI:10.1161/CIRCRESAHA.120.318501
PMID:33902304
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8221541/
Abstract

[Figure: see text].

摘要

[图：见正文]。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/d3dc3de3fbb6/res-129-63-g007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/74bb197ace98/res-129-63-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/ba6652847d59/res-129-63-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/e441e51737e2/res-129-63-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/6761ad55fd34/res-129-63-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/1df7c3cdf983/res-129-63-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/f5a76299d08b/res-129-63-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/d3dc3de3fbb6/res-129-63-g007.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/74bb197ace98/res-129-63-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/ba6652847d59/res-129-63-g002.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/e441e51737e2/res-129-63-g003.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/6761ad55fd34/res-129-63-g004.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/1df7c3cdf983/res-129-63-g005.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/f5a76299d08b/res-129-63-g006.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/7c93/8221541/d3dc3de3fbb6/res-129-63-g007.jpg

相似文献

[1]
Prdm16 Supports Arterial Flow Recovery by Maintaining Endothelial Function.

Circ Res. 2021-6-25

[2]
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[3]
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[4]
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[5]
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[6]
Deletion of FHL2 gene impaired ischemia-induced blood flow recovery by modulating circulating proangiogenic cells.

Arterioscler Thromb Vasc Biol. 2013-2-14

[7]
Requirement of β1 integrin for endothelium-dependent vasodilation and collateral formation in hindlimb ischemia.

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[8]
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[9]
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[10]
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引用本文的文献

[1]
PRDM16, a new kid on the block in cardiovascular health and disease.

Cardiovasc Res. 2025-7-31

[2]
Factors associated with blood pressure regulation in vascular smooth muscle cells.

Hypertens Res. 2025-5

[3]
PRDM16 controls smooth muscle cell fate in atherosclerosis.

bioRxiv. 2025-2-23

[4]
PRDM16 determines specification of ventricular cardiomyocytes by suppressing alternative cell fates.

Life Sci Alliance. 2024-12

[5]
PBX/Knotted 1 homeobox-2 (PKNOX2) is a novel regulator of myocardial fibrosis.

Signal Transduct Target Ther. 2024-4-22

[6]
Epigenetic signature of human immune aging in the GESTALT study.

Elife. 2023-8-17

[7]
PRDM16 regulates arterial development and vascular integrity.

Front Physiol. 2023-6-1

[8]
PRDM16 deficiency in vascular smooth muscle cells aggravates abdominal aortic aneurysm.

JCI Insight. 2023-6-8

[9]
Angiotensin receptor-neprilysin inhibitor improves coronary collateral perfusion.

Front Cardiovasc Med. 2023-2-3

[10]
A cardiac-null mutation of Prdm16 causes hypotension in mice with cardiac hypertrophy via increased nitric oxide synthase 1.

PLoS One. 2022

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