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Netrin-1 marshals mitochondrial movement, morphology, and metabolism in myelin.

作者信息

Nakamura Diane S, Kennedy Timothy E

机构信息

Montreal Neurological Institute, Department of Neurology and Neurosurgery, McGill University, Montreal, QC, Canada.

出版信息

Neural Regen Res. 2021 Dec;16(12):2399-2400. doi: 10.4103/1673-5374.313028.

DOI:10.4103/1673-5374.313028
PMID:33907016
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC8374595/
Abstract
摘要
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9d14/8374595/01f27de7d0aa/NRR-16-2399-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9d14/8374595/01f27de7d0aa/NRR-16-2399-g001.jpg
https://cdn.ncbi.nlm.nih.gov/pmc/blobs/9d14/8374595/01f27de7d0aa/NRR-16-2399-g001.jpg

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本文引用的文献

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Mitochondrial dynamics and bioenergetics regulated by netrin-1 in oligodendrocytes.少突胶质细胞中轴突导向因子 netrin-1 对线粒体动力学和生物能量的调控
Glia. 2021 Feb;69(2):392-412. doi: 10.1002/glia.23905. Epub 2020 Sep 10.
2
High-Frequency Microdomain Ca Transients and Waves during Early Myelin Internode Remodeling.高频微域钙瞬变和波在早期髓鞘节段重塑过程中的作用。
Cell Rep. 2019 Jan 2;26(1):182-191.e5. doi: 10.1016/j.celrep.2018.12.039.
3
Activity-Dependent Netrin-1 Secretion Drives Synaptic Insertion of GluA1-Containing AMPA Receptors in the Hippocampus.
髓鞘中的线粒体募集：髓鞘动态变化和可塑性的支撑？
Neural Regen Res. 2024 Jul 1;19(7):1401-1402. doi: 10.4103/1673-5374.387982. Epub 2023 Nov 8.
4
Identifying mRNAs Residing in Myelinating Oligodendrocyte Processes as a Basis for Understanding Internode Autonomy.鉴定存在于髓鞘形成少突胶质细胞突起中的mRNA，作为理解结间体自主性的基础。
Life (Basel). 2023 Apr 4;13(4):945. doi: 10.3390/life13040945.
活动依赖性 Netrin-1 分泌驱动海马体中 GluA1 包含的 AMPA 受体的突触插入。
Cell Rep. 2018 Oct 2;25(1):168-182.e6. doi: 10.1016/j.celrep.2018.09.028.
4
Inhibition of Drp1 hyper-activation is protective in animal models of experimental multiple sclerosis.抑制动力相关蛋白1（Drp1）的过度激活在实验性多发性硬化症动物模型中具有保护作用。
Exp Neurol. 2017 Jun;292:21-34. doi: 10.1016/j.expneurol.2017.02.015. Epub 2017 Feb 24.
5
Movement and structure of mitochondria in oligodendrocytes and their myelin sheaths.少突胶质细胞及其髓鞘中线粒体的运动与结构
Glia. 2016 May;64(5):810-25. doi: 10.1002/glia.22965. Epub 2016 Jan 17.
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Progressive disorganization of paranodal junctions and compact myelin due to loss of DCC expression by oligodendrocytes.少突胶质细胞中 DCC 表达缺失导致连接结和髓鞘板层的进行性解体。
J Neurosci. 2014 Jul 16;34(29):9768-78. doi: 10.1523/JNEUROSCI.0448-14.2014.
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Cell. 2014 Jan 16;156(1-2):277-90. doi: 10.1016/j.cell.2013.11.044.
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