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迟发性波特氏截瘫。压迫原因及前路减压后的结果。

Pott's paraplegia of late onset. The cause of compression and results after anterior decompression.

作者信息

Hsu L C, Cheng C L, Leong J C

机构信息

Department of Orthopaedic Surgery, University of Hong Kong, Queen Mary Hospital.

出版信息

J Bone Joint Surg Br. 1988 Aug;70(4):534-8. doi: 10.1302/0301-620X.70B4.3403593.

DOI:10.1302/0301-620X.70B4.3403593
PMID:3403593
Abstract

Twenty-two patients with late onset Pott's paraplegia presenting at a mean of 18 years after initial symptoms were reviewed an average of seven years after treatment by anterior decompression and fusion. Fourteen patients had active disease, and in 12 of these, activity at the internal kyphus was the direct cause of the paraplegia. In the other two, a soft healing bony ridge was the cause. The eight patients with healed disease had hard bony ridges compressing the cord. The response to anterior decompression was faster, better and safer in patients with active disease: nine recovered completely and three significantly. In patients with healed disease, the anterior decompression was technically more difficult and the recovery less satisfactory. Significant complications included two cases with neurological deterioration, two with cerebrospinal fluid fistulae and four with neurapraxia of the cord.

摘要

对22例迟发性波特氏截瘫患者进行了回顾性研究,这些患者在出现初始症状后平均18年就诊,接受前路减压融合治疗后平均随访7年。14例患者存在活动性疾病,其中12例患者胸段后凸部位的病变活动是截瘫的直接原因。另外2例患者,截瘫是由愈合中的软质骨嵴所致。8例疾病已愈合的患者存在坚硬的骨嵴压迫脊髓。活动性疾病患者接受前路减压后反应更快、更好且更安全:9例完全恢复,3例有显著改善。疾病已愈合的患者,前路减压在技术上更困难,恢复情况也不太令人满意。严重并发症包括2例神经功能恶化、2例脑脊液漏和4例脊髓神经失用。

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