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Corrigendum to "miR-221/222 promote cancer stem-like cell properties and tumor growth of breast cancer via targeting PTEN and sustained Akt/NF-κB/COX-2 activation" [Chem. Biol. Interact. 277 (2017) 33-42].

作者信息

Li Bailong, Lu Ying, Yu Lihui, Han Xiaocui, Wang Honghai, Mao Jun, Shen Jie, Wang Bo, Tang Jianwu, Li Chunyan, Song Bo

机构信息

Department of Pathology and Forensics, Dalian Medical University, No.9 West Section, Lvshun South Road, Dalian, 116044, China; Department of Clinical Medicine, Grade 2013, Dalian Medical University, No. 9 West Section, Lvshun South Road, Dalian, 116044, China.

Teaching Laboratory of Morphology, Dalian Medical University, No.9 West Section, Lvshun South Road, Dalian, 116044, China.

出版信息

Chem Biol Interact. 2021 Aug 25;345:109556. doi: 10.1016/j.cbi.2021.109556. Epub 2021 Jun 29.

DOI:10.1016/j.cbi.2021.109556
PMID:34210408
Abstract
摘要

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Corrigendum to "miR-221/222 promote cancer stem-like cell properties and tumor growth of breast cancer via targeting PTEN and sustained Akt/NF-κB/COX-2 activation" [Chem. Biol. Interact. 277 (2017) 33-42].对“miR-221/222通过靶向PTEN和持续激活Akt/NF-κB/COX-2促进乳腺癌干细胞样特性和肿瘤生长”[《化学生物学相互作用》277 (2017) 33 - 42]的勘误
Chem Biol Interact. 2021 Aug 25;345:109556. doi: 10.1016/j.cbi.2021.109556. Epub 2021 Jun 29.
2
miR-221/222 promote cancer stem-like cell properties and tumor growth of breast cancer via targeting PTEN and sustained Akt/NF-κB/COX-2 activation.miR-221/222 通过靶向 PTEN 并持续激活 Akt/NF-κB/COX-2 促进乳腺癌肿瘤干细胞样特性和肿瘤生长。
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Nuclear factor-κB-dependent microRNA-130a upregulation promotes cervical cancer cell growth by targeting phosphatase and tensin homolog.核因子-κB依赖性微小RNA-130a上调通过靶向磷酸酶和张力蛋白同源物促进宫颈癌细胞生长。
Arch Biochem Biophys. 2016 May 15;598:57-65. doi: 10.1016/j.abb.2016.03.019. Epub 2016 Apr 1.
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miR-10b expression in breast cancer stem cells supports self-renewal through negative PTEN regulation and sustained AKT activation.乳腺癌干细胞中的miR-10b表达通过负向调控PTEN和持续激活AKT来支持自我更新。
EMBO Rep. 2016 May;17(5):648-58. doi: 10.15252/embr.201540678. Epub 2016 Apr 9.
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Her-2/neu overexpression induces NF-kappaB via a PI3-kinase/Akt pathway involving calpain-mediated degradation of IkappaB-alpha that can be inhibited by the tumor suppressor PTEN.Her-2/neu过表达通过PI3激酶/Akt途径诱导核因子κB，该途径涉及钙蛋白酶介导的IkappaB-α降解，而这一过程可被肿瘤抑制因子PTEN抑制。
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