Forsander O A, Pösö A R
Research Laboratories of the State Alcohol Company (Alko, Ltd), Helsinki, Finland.
Alcohol Alcohol Suppl. 1987;1:357-9.
Ethanol increased significantly the rate of gluconeogenesis from alanine in rats from the AA strain (alcohol-preferring animals), while no change was observed in rats from the ANA strain (alcohol-avoiding animals). The glycogen stores after alcohol administration were depleted in the livers of the ANA rats but remained unaffected in the AA animals. The results indicate that alcohol disturbs the carbohydrate homeostatis in the ANA rats, but not in the AA animals.
乙醇显著提高了 AA 品系（偏好酒精的动物）大鼠由丙氨酸生成葡萄糖的速率，而 ANA 品系（回避酒精的动物）大鼠未观察到变化。给予酒精后，ANA 大鼠肝脏中的糖原储备减少，而 AA 大鼠的糖原储备未受影响。结果表明，酒精扰乱了 ANA 大鼠的碳水化合物稳态，但对 AA 大鼠没有影响。
